The chart below lists usage by number of pharmacy claims of the agents included in this review between 7 01 04 and 7 01 05. Generic Name AMLODIPINE ATORVASTATIN AMLODIPINE BENAZEPRIL ENALAPRIL FELODIPINE TRANDOLAPRIL VERAPAMIL TOTAL Rx Num 8, 775 87, As an annual review, this document is intended to include only products or product information not available at the time of the original review of this class. No new products have entered the marketplace since these agents were last reviewed. Also, no new product information has been made available regarding pharmacology, pharmacokinetics, drug interactions, adverse effects, dosage and administration or efficacy.
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6must be returned immediately to the Company. However, the Committee may provide for partial or complete exceptions to this requirement as it deems equitable. b ; Restrictions on Transfer and Legend on Stock Certificate. During the Restriction Period, a Grantee may not sell, assign, transfer, pledge, or otherwise dispose of the shares of Lilly Stock except to a Successor Grantee under Section 10 a ; . Each certificate for shares issued or transferred under a Restricted Stock Grant shall contain a legend giving appropriate notice of the restrictions in the Grant. c ; Lapse of Restrictions. All restrictions imposed under the Restricted Stock Grant shall lapse upon the expiration of the Restriction Period if all conditions stated in Sections 8 a ; and b ; have been met. The Grantee shall then be entitled to have the legend removed from the certificate.
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If symptoms present for 2 years, Chronic Depression, or functional impairment is severe, remission with watchful waiting is unlikely, immediate active treatment indicated for moderate depressive symptoms minor depression ; . * Referral or co-management with mental health specialty clinician if patient is a high suicide risk or has bipolar disorder, an inadequate treatment response, or complex psychosocial needs and or other active mental disorders Using PHQ-9 for Diagnostic Assessment Of the 9 items in question 1, include only those that are checked at least "More than half the days", except count the suicide item if present "at all" At least one of item 1a or item 1b must be endorsed as more than half the days for a depression diagnosis. Also question 2 for functional impairment must be 3 answered at least "Somewhat difficult." Using PHQ-9 For Severity of Depression Measure Of the 9 items in question 1, also include items checked "Several days." Count one point for each item checked several days, two points for checked items more than half the days, three points for items checked nearly every day, and sum the total for a severity score.
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Note Differences in the classifications of biotech products, the connection of producing companies to pharmaceutical research and other institutions, and the small size of the many start-up companies make obtaining precise and comparable statistics difficult. This report employs Ernst & Young's definition of biotechnology and the statistical data, since the firm's reports on the state of biotechnology in Germany are widely accepted as standard. According to Ernst & Young biotechnology includes all innovative methods, products and treatments containing living organisms or their cellular parts, as well as, all commercial transfer of knowledge from the fields of molecular biology, virology, and microbiology and cell biology. Products and treatments that are not purely life science, but that play a significant role in the biotech industry, such as bio-informatics and combinatorial analysis, also fall in this category. The focus is on "core biotech firms" whose aim is the commercialization of modern biotech products, technologies and services. ; Section 1-1. Market Overview Over the past eight years, Germany became the fastest growing biotechnology market in Europe: 21 % of Europe's biotech companies are located in Germany. Nevertheless, since 2002 the German biotech industry has been facing economic difficulties, similar to other global industries. Table I: The German Biotechnology Industry.
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26 Yaney, G. C., Korchak, H. M. and Corkey, B. E. 2000 ; Long-chain acyl CoA regulation of protein kinase C and fatty acid potentiation of glucose-stimulated insulin secretion in clonal -cells. Endocrinology 141, 19891998 27 Longo, E. A., Tornheim, K., Deeney, J. T., Varnum, B. A., Tillotson, D., Prentki, M. and Corkey, B. E. 1991 ; Oscillations in cytosolic free Ca2 + , oxygen consumption, and insulin secretion in glucose-stimulated rat pancreatic islets. J. Biol. Chem. 266, 93149319 28 Cunningham, B. A., Deeney, J. T., Bliss, C. R., Corkey, B. E. and Tornheim, K. 1996 ; Glucose-induced oscillatory insulin secretion in perifused rat pancreatic islets and clonal -cells HIT ; . Am. J. Physiol. 271, E702E710 29 Gotoh, M., Maki, T., Satomi, S. J. P., Bonner-Weir, S., O 'Hara, C. J. and Monaco, A. P. 1987 ; Reproducible high yield of rat islets by stationary in vitro digestion following pancreatic ductal or portal venous collagenase injection. Transplantation 43, 725730 30 Darville, M. I., Chikri, M., Lebeau, E., Hue, L. and Rousseau, G. G. 1991 ; A rat gene encoding heart 6-phosphofructo-2-kinase fructose-2, 6-bisphosphatase. FEBS Lett. 288, 9194 31 Veldhuis, J. D. and Johnson, M. L. 1986 ; Cluster analysis : a simple, versatile, and robust algorithm for endocrine pulse detection. Am. J. Physiol. 250, E486E493 32 Hermier, D., Hales, P. and Brindley, D. N. 1991 ; Effects of the lipase inhibitors, Triton WR-1339 and tetrahydrolipstatin, on the synthesis and secretion of lipids by rat hepatocytes. FEBS Lett. 286, 186188 33 Krebs, M., Stingl, H., Nowotny, P., Weghuber, D., Bischof, M., Waldhausl, W. and Roden, M. 2000 ; Prevention of in vitro lipolysis by tetrahydrolipstatin. Clin. Chem. 46, 950954 34 Smith, G. M., Garton, A. J., Aitken, A. and Yeaman, S. J. 1996 ; Evidence for a multidomain structure for hormone-sensitive lipase. FEBS Lett. 396, 9094 35 Cunningham, B. A., Tornheim, K. and Corkey, B. E. 1995 ; Cyclic AMP alters oscillatory insulin secretion. Diabetologia 38, A104 36 Porksen, N., Grofte, B., Nyholm, B., Holst, J. J., Pincus, S. M., Veldhuis, J. D., Schmitz, O. and Butler, P. C. 1998 ; Glucagon-like peptide 1 increases mass but not frequency or orderliness of pulsatile insulin secretion. Diabetes 47, 4549 37 Porksen, N., Munn, S., Steers, J., Veldhuis, J. D. and Butler, P. C. 1996 ; Effects of glucose ingestion versus infusion on pulsatile insulin secretion. The incretin effect is achieved by amplification of insulin secretory burst mass. Diabetes 45, 13171323 38 Baltrusch, S., Lenzen, S., Okar, D. A., Lange, A. J. and Tiedge, M. 2001 ; Characterization of glucokinase-binding protein epitopes by a phage-displayed peptide library. Identification of 6-phosphofructo-2-kinase fructose-2, 6-bisphosphatase as a novel interaction partner. J. Biol. Chem. 276, 4391543923 39 Berne, C. 1975 ; The metabolism of lipids in mouse pancreatic islets. The biosynthesis of triacylglycerols and phospholipids. Biochem. J. 152, 667673 40 Vara, E. and Tamarit-Rodriguez, J. 1986 ; Glucose stimulation of insulin secretion in islets of fed and starved rats and its dependence on lipid metabolism. Metab. Clin. Exp. 35, 266271 41 Corkey, B. E., Deeney, J. T., Yaney, G. C., Tornheim, K. and Prentki, M. 2000 ; The role of long-chain fatty acyl-CoA esters in -cell signal transduction. J. Nutr. 130, 299S304S 42 Masiello, P., Novelli, M., Bombara, M., Fierabracci, V., Vittorini, S., Prentki, M. and Bergamini, E. 2002 ; The antilipolytic agent 3, 5-dimethylpyrazole inhibits insulin release in response to both nutrient secretagogues and cyclic adenosine monophosphate agonists in isolated rat islets. Metab. Clin. Exp. 51, 110114 43 Gromada, J., Holst, J. J. and Rorsman, P. 1998 ; Cellular regulation of islet hormone secretion by the incretin hormone glucagon-like peptide 1. Pflugers Arch. Eur. J. Physiol. 435, 583594 44 Renstro$ m, E., Eliasson, L. and Rorsman, P. 1997 ; Protein kinase A-dependent and -independent stimulation of exocytosis by cAMP in mouse pancreatic B-cells. J. Physiol. Cambridge, U.K. ; 502, 105118 45 Gromada, J., Ding, W. G., Barg, S., Renstro$ m, E. and Rorsman, P. 1997 ; Multisite regulation of insulin secretion by cAMP-increasing agonists : evidence that glucagon-like peptide 1 and glucagon act via distinct receptors. Pflugers Arch. Eur. J. Physiol. 434, 515524 46 Kashima, Y., Miki, T., Shibasaki, T., Ozaki, N., Miyazaki, M., Yano, H. and Seino, S. 2001 ; Critical role of cAMP-GEFII Rim2 complex in incretin-potentiated insulin secretion. J. Biol. Chem. 276, 4604646053 47 Roduit, R., Masiello, P., Wang, S. P., Li, H., Mitchell, G. A. and Prentki, M. 2001 ; A role for hormone-sensitive lipase in glucose-stimulated insulin secretion : A study in hormone-sensitive lipase-deficient mice. Diabetes 50, 19701975 48 Antinozzi, P. A., Segall, L., Prentki, M., McGarry, J. D. and Newgard, C. B. 1998 ; Molecular or pharmacologic perturbation of the link between glucose and lipid metabolism is without effect on glucose-stimulated insulin secretion. A re-evaluation of the long-chain acyl-CoA hypothesis. J. Biol. Chem. 273, 1614616154.
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This presentation has been organised by Alizyme plc the "Company" ; in order to provide general information on the Company. This material has been prepared solely by the Company and is i ; for your private information, and the Company is not soliciting any action based upon it; ii ; not to be construed as an offer to sell or a solicitation of an offer to buy any security and iii ; based upon information that the Company considers reliable. The Company does not represent that the information contained in this material is accurate or complete, and it should not be relied upon as such. No representation, warranty or undertaking, express or implied, is or will be made with respect to the fairness, accuracy or completeness of any of the information or statement of opinion or expectation contained herein or stated in the presentation or any other such information nor shall you be entitled to rely upon it. In furnishing you with this information no obligation is undertaken to provide you with any further information, to update this information nor any other information nor to correct any information contained herein or any omission therefrom. The Company's securities have not been registered under the U.S. Securities Act of 1933 as amended ; , and may not be offered or sold in the United States or to U.S. persons unless they have been registered under such Act, or except in compliance with an exemption from the registration requirements of such Act. No part of this material may be i ; copied, photocopied, or duplicated in any form, by any means, or ii ; redistributed, published, or disclosed by recipients to any other person, in each case without the Company's prior written consent. This material is only being provided to persons who are authorised persons or exempted persons within the meaning of the Financial Services and Markets Act 2000 or any order made thereunder or to other persons of a kind described in Articles 19 and 49 of the Financial Services and Markets Act 2000 Financial Promotions ; Order 2005 or who are otherwise permitted by law to receive it. In relation to information about the price at which securities in the Company have been bought or sold in the past, note that past performance cannot be relied upon as a guide to future performance. In addition, the occurrence of some of the events described in this document and the presentation that will be made, and the achievement of the intended results, are subject to the future occurrence of many events, some or all of which are not predictable or within the Company's control; therefore, actual results may differ materially from those anticipated in any forward looking statements. The Company disclaims any obligation to update these forward looking statements. The financial information does not constitute statutory financial statements within the meaning of section 240 of the Companies Act 1985. The results for the period ended 30 June 2007 has been extracted from the Interim Report, which has been reviewed in accordance with the guidance contained in Bulletin 1999 4 issued by the Auditing Practices Board for use in the United Kingdom. The results for the periods ended 31 December 2006, 31 December 2005 and 31 December 2004 have been extracted from the statutory financial statements from each year, which have been filed with the Registrar of Companies in England and Wales and upon which the auditors reported without qualification.
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