Reducing stigma, improving provider attitudes, ensuring confidentiality, and promoting and providing benefits to both HIV-positive and HIV-negative women. 3. Promote rapid testing, without confirmatory testing before informing the client of her status, and social marketing of VCT. 4. Establish guidelines for health workers and trained counselors in every facility for counseling HIV-positive pregnant women about decreasing the chances of MTCT through infant feeding options. Always observe their human rights and support their choices in all aspects of HIVpositive women's own health and that of their babies. 5. Guarantee that family planning and health and counseling services are universally available to maximize the HIV-positive woman's chances of preventing pregnancy or avoiding transmitting the virus to her baby, to support her in her choice of feeding method materially and emotionally, and to provide care to a baby.
And unresponsive to a single diuretic. These medications may not be necessary if the patient responds to activity recommendations, avoidance of excessive fluid intake 2 quarts day ; , and a lowsodium diet eg, 2 g day ; . Spironolactone Aldacton4 ; is a potassium-sparing diuretic that inhibits sodium reabsorption in the late distal tubule and collecting duct. It has been found to be effective in reducing mortality and morbidity in NYHA class III and IV HF patients when added to ACE-Is, loop diuretics, and digoxin. Serum creatinine and potassium levels are monitored frequently eg, within the first week and then every 4 weeks ; when this medication is first administered. Side effects of diuretics include electrolyte imbalances, symptomatic hypotension especially with overdiuresis ; , hyperuricemia causing gout ; , and ototoxicity. Dosages depend on the indications, patient age, clinical signs and symptoms, and renal function. Table 30-4 lists commonly used diuretics, dosages, and pharma.
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11. Chobanian A, Bakris G, Black H, et al. Seventh Report of The Joint National Committee on Prevention, Detection, Evaluation, and Management of High Blood Pressure JNC-7 ; . Hypertension 2003; 42: 12061252. Hunt S, Abraham WT, Chin M, et al. ACC AHA 2005 Guideline Update for the Diagnosis and Management of Chronic Heart failure in the Adult. A report of the American College of Cardiology American Heart Association task force on practice guidelines. Writing to update the 2001 guidelines for the evaluation and management of heart failure, 2005, available at acc , accessed 10-19-05. 13. Dargie HJ. Effect of carvedilol on outcome after myocardial infarction in patients with left-ventricular dysfunction: the CAPRICORN randomized trial. Lancet 2001; 357: 13851390. Packer M, Coats A, Fowler MB, et al; Carvedilol Prospective Randomized Cumulative Survival Study Group. Effect of carvedilol on survival in severe chronic heart failure. N Engl J Med 2001; 344: 16511658. Effect of metoprolol CR XL in chronic heart failure: Metoprolol CR XL Randomized Intervention Trial in Congestive Heart Failure MERIT-HF ; . Lancet 1999, 353: 20012007. Pitt B, Zannad F, Remme WJ, et al. The effect of spironolactone on morbidity and mortality in patents with severe heart failure. Randomized Aldactoe Evaluation Study RALES ; Investigators. N Engl J Med 1999; 341: 709717. Pitt B, Remme WJ, Zannad F, et al; Eplerenone PostAcute Myocardial Infarction Heart Failure Efficacy and Survival EPHESUS ; Study Investigators. Eplerenone, a selective aldosterone blocker, in patients with left ventricular dysfunction after myocardial infarction. N Engl J Med 2003; 14: 13091321. Beta-Blocker Heart Attack BHAT ; Research Group. A randomized trial of propranalol in patients with acute myocardial infarction, I: mortality results. JAMA 1982; 247: 17071714. Timolol-induced reduction in mortality and reinfarction in patients surviving acute myocardial infarction. N Engl J Med 1981; 304: 801807. Nissen SE, Tuzcu EM, Libby P, et al. Effect of antihypertensive agents on cardiovascular events in patients with coronary disease and normal blood pressure. The CAMELOT Study: a randomized controlled trial. JAMA 2004; 292: 22172226.
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Fatal EBV Infection disclosed a prothrombin time of 19.4 sec with a control value of 10.1 sec, a partial thromboplastin time of 50 sec with a control value of 24.4 sec, a thrombin time of 30.6 sec with a control value of 20.5 sec, and a fibrinogen value of 40 units. Fibrin degradation products were not significantly elevated. His bone marrow was hypercellular with increased numbers of mature histiocytes, some demonstrating erythrophagocytosis. He was treated with spironolactone Aldactone; G. D. Bearle and Co., Chicago, III. ; , i.v. albumin, and prednisone, 40 mg sq m day for 1 week. On November 21, 1979, while receiving prednisone, he developed recurrent fever 38 ; and had an increase in spleen size to 15 cm below the right costal margin. Prednisone therapy was gradually decreased and finally stopped altogether. His clinical condition continued to deteriorate with high fevers daily, massive splenomegaly 15 cm below the right costal margin, and bilateral cervical lymphadenopathy with nodes measuring 2x4 cm. Laboratory studies at that time disclosed a hemoglobin of 7 g despite daily transfusions, a WBC of 900 30% polymorphonuclear leukocytes, 60% lymphocytes, and 10% monocytes ; , and a platelet count of 8000 cu mm. His bone marrow contained normal hematopoietic precursors, as well as transformed lymphocytes and numerous histiocytes demonstrating erythrophagocytosis. The uric acid concentra tion was 13.8 mg dl, and serum mmunoglobulin studies showed an IgG of 2100 mg dl normal range, 650 to 1600 mg dl ; , an IgA of 330 mg dl normal range, 100 to 400 mg dl ; , and an IgM of 1500 mg dl normal range, 18 to 280 mg dl ; , with a polyclonal pattern of IgM by immunoelectrophoresis. On December 4, 1979, a lymph node biopsy was obtained, and i.v. total parenteral nutrition was begun. From December 4 to 12, the patient continued to have high fever, although his spleen and cervical lymph nodes decreased spontaneously in size. His pancytopenia progressed during this period, and on December 12, he had 300 WBCs with no polymorphonuclear leukocytes on smears, a hemoglobin of 8.5 mg dl transfused ; , and no detectable platelets on smears. A bone marrow aspirate was extremely hypocellular. From December 12 to 19, his high fever persisted; he developed progressive cardiomegaly, pul monary edema, and cardiorespiratory failure, which led to his death on December 19. Results Pathological Findings. In November 1979, a bone marrow biopsy disclosed that the patient's marrow was hypercellular with panhyperplasia and a shift towards immaturity in the myeloid and erythroid series. Most importantly, a significant histiocytic infiltration was recognized. The cytologically bland histiocytes were phagocytic for erythrocytes, platelets, and rarely leukocytes. In light of the clinical and viral serological features, the histopathological findings were consistent with a virally induced hemophagocytic syndrome 19 ; . No fungi were noted in the sections, smears, or cultures. When massive lymphadenopathy developed during the next few weeks, a cervical lymph node was excised. The nodal architecture was diffusely effaced by a proliferation of lymphoid cells showing a wide spectrum of maturation, ranging from atypical small lymphocytes to immunoblasts. The histological features seen in this section were not those of any of the wellNOVEMBER 1981 characterized malignant lymphomas but were those been observed in other cases of EBV infections. The potential of this atypical immunoblastic proliferation tain. In the ensuing 7 to 10 days, the patient's condition that have malignant is uncer changed and aldara.
Note to existing members: This formulary has changed since last year. Please review this document to make sure that it still contains the drugs you take. This document includes AbilityCare's partial formulary as of January, 2007. For a complete, updated formulary, please visit our Web site at mnscha or call 1-866-477-1601, 7 days a week, 8: 00 a.m. to 8: 00 p.m. TTY TDD users should call 1-888-878-0137.
Bipolar research today home view latest issue information about bipolar books on bipolar view other research today publications chronic nmda administration to rats up-regulates frontal cortex cytosolic phospholipase a 2 ; and its transcription factor, activator protein- rao js, ertley rn, rapoport si, bazinet rp, lee hj brain physiology and metabolism section, national institute on aging, national institutes of health, bethesda, maryland, usa excessive n-methyl-d-aspartate nmda ; signaling is thought to contribute to bipolar disorder symptoms and alendronate, because aldactone interactions.
| Aldactone dosage for hirsutismAll i was saying is that medical professionals follow a procedure when it comes to diagnosis and that sometimes you end up with someone not worth their salt.
Aldactone is a diuretic used for the shedding of excess water, particularly before a sho inserts they would know this and amlodipine.
The investigator is required to report all treatment-emergent adverse events that are observed or reported during study phase 2 i.e., treatment phase ; . This applies regardless of a ; the clinical significance or b ; the assessment of study drug causality. All such events must be recorded on the AEF. An adverse event is defined as any untoward medical occurrence in a patient who takes the study medication. It does not necessarily have to have a causal relationship with this treatment. This may be any unfavorable and unintended sign e.g., abnormal laboratory finding ; , symptom or disease, which is observed after exposure to the study medication, whether or not considered, related to the treatment.
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O'Brien JD, Howlett SE Department of Pharmacology, Dalhousie University, Halifax, Canada Corresponding Author: jdobrien dal Funding Source: CIHR, HSFNS Background: Cardioprotective effects of ischemic preconditioning IPC ; are thought to decrease with age. We compared the impact of age on contraction, Ca2 + homeostasis and cell survival in cardiac myocytes exposed to IPC prior to prolonged ischemia and reperfusion. Methods: Myocytes from young adult 3 mos ; and aged 24 mos ; male Fischer 344 rats were field stimulated at 4 Hz 37C ; . Contraction and intracellular Ca2 + were measured with an edge detector and fura 2. Cells were preconditioned with 5 mins of simulated ischemia prior to prolonged ischemia 30 mins ; and were then reperfused with Tyrode's solution. Results: IPC abolished post-ischemic contractile dysfunction and increased Ca2 + transient amplitudes throughout reperfusion in young adult cells. In contrast, IPC did not improve contractile function and Ca2 + transients until late reperfusion in aged cells. Further, IPC improved cell survival in reperfusion in young adult but not aged myocytes. Despite these protective effects, IPC had little effect on the brief rise in diastolic Ca2 + during ischemia in young adult cells, although IPC attenuated the marked increase in diastolic Ca2 + in ischemia in aged cells. Conclusions: IPC improves contractile function and cell survival in young adult myocytes, but is much less effective in aged myocytes. These protective effects are not mediated by a decrease in diastolic Ca2 + in ischemia, at least in young adult myocytes. This decrease in effectiveness of IPC at the level of the myocyte may increase sensitivity of aging heart to ischemic heart disease. Keywords: Aging, ischemia reperfusion, intracellular calcium and clavulanate.
[Mary Coughlan.] moted through agricultural education and training. My Department closely monitors ongoing research programme both on climate change in Ireland and its likely impact. I aware that increased precipitation and water shortages are major issues for agriculture worldwide and it has been suggested that such changes in our own climate may have both positive and negative long-term effects on Irish agriculture. Accordingly, my Department is funding various research projects to assist in identifying sustainable greenhouse gas emission reduction measures. Amongst these are studies focused on reducing methane emissions in ruminants and an examination of nitrous oxide emissions from grasslands. I conscious of the commitments made in the National Development Plan to combat climate change and I intend, through my Department's network of local offices, the offices of Teagasc and by providing support to relevant NGOs, to pursue a campaign to increase awareness of, and the need for adaptation to, the impacts of climate change in the agriculture and forestry sectors. Mushroom Industry. 110. Mr. G. Mitchell asked the Minister for Agriculture and Food the inspection regimes administered by her Department in relation to the mushroom growing industry; the frequency with which such inspections take place; the penalties in place for failure to comply with these Departmental regulations; the number of such establishments found to be in breach of existing regulations in 2005 and 2006; and if she will make a statement on the matter. [3549 07] Minister for Agriculture and Food Mary Coughlan ; : The regulatory framework governing plant protection products in Ireland which is set out in SI 83 2003 is designed to ensure a very high standard of protection for human health and the environment. Enforcement of the legislation involves inspections to ensure that only approved products are present in the market and are used by farmers and growers. Inspections normally take place at wholesale distribution level. However where there is evidence of possible misuse of plant protection products generated through the residue monitoring programme at wholesale distribution level or from any other source, specific inspections at end-user level take place. The pesticide residue monitoring programme conducted by my Department on behalf of the Food Safety Authority of Ireland FSAI ; , is agreed on an annual basis with the FSAI. This programme is risk-based and involved the analysis of some 1, 350 samples of agricultural produce in each of the years 2005 and 2006 for up to 150 different pesticide compounds. The number of, for example, aldactone ascites.
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Important in mediating the improvement in ejection fraction after -blocker treatment. However, even patients with no hibernating myocardium should be commenced on -blocker therapy, because -blockers are likely to have effects beyond improvement in LV function, such as suppression of lethal ventricular tachyarrhythmias. REVASCULARIZATION FOR HEART FAILURE In contrast to CHRISTMAS, which examined the interaction between myocardial hibernation and medical therapy, there are many trials examining the interaction between myocardial hibernation and revascularization. The most comprehensive review is the meta-analysis by Allman et al, 9 which summarizes 24 trials performed between 1966 and August 1999. Among them, these trials encompassed all of the clinically available techniques for identifying hibernation. Three thousand eighty-eight patients were included in the meta-analysis. The mean ejection fraction was 32% 8%. In patients with viability, revascularization was associated with a striking 80% reduction in the annual mortality rate from 16% to 3.2% ; compared with medical treatment alone. The benefit observed was directly related to the severity of LV dysfunction, but in the absence of viability, no benefit of revascularization was found at any level of ejection fraction. The meta-analysis did not include the quantitative relationship between extent of myocardial viability hibernation and improved outcome after revascularization. However, individual studies have attempted to address this issue. Most studies suggest that between one third to one half of total myocardial mass needs to be affected before a survival benefit from revascularization is seen.10 In interpreting these data, it is important to note that since the meta-analysis was conducted, medical therapy has advanced. For example, in addition to angiotensinconverting enzyme inhibitors, spironolactone and -blockers have been shown to be effective for heart failure. In the RALES trial Randomized Aldactne Evaluation Study ; , published in 1999, there was a 30% reduction in mortality rate.11 In 1996 the US Carvedilol Program results first clearly showed the benefit of -blocker therapy, a finding that was subsequently confirmed by the CIBIS-II Cardiac Insufficiency Bisoprolol Study II ; 12 and MERIT-HF Metoprolol CR XL Randomized Intervention Trial in congestive heart failure ; trials.13 These were published in 1999 and 2000, respectively, and both demonstrated a reduction of 30% to 35% in the mortality rate. As neither spironolactone nor -blockers were widely used for the treatment of heart failure before 1999, this still leaves open the question of whether.
Our target for the current term are to speed up the drug R&D process right up to application for approval and to enrich our current R&D pipelines. In the field of new drug discovery, we are investing management resources in a particularly focused manner in the therapeutic areas of urology, and inflammation allergies. In addition, we are working to license-in new drugs from other companies in the aforementioned two therapeutic areas as well as in blood malignancies. Regarding in-house-developed drugs that are competitive in overseas markets, we will be making efforts to market such drugs in various countries through the licensingout as well as joint development. To realize these goals, we will selectively focus management resources on the most promising drug candidates and target, for example, aldactone generic.
Substance or its Metabolites or Markers ; , Regulation 21.2.2 Use or Attempted Use of Prohibited Substance or Prohibited Method ; and Regulation 21.2.6 Possession of Prohibited Substances and Methods ; shall be: First violation: Two 2 ; years' Ineligibility. Second violation: Lifetime Ineligibility. However, the Player or other Person shall have the opportunity in each case, before a period of Ineligibility is imposed, to establish the basis for eliminating or reducing this sanction as provided in Regulation 21.22.4. 33. It was confirmed that this is the Player's first anti-doping rule violation. 34. Regulation 21.22.4 contains provisions for the elimination or reduction of a period of Ineligibility based on "Exceptional Circumstances". 35. Two categories of exceptional circumstances are identified. In the first, if a Player can establish that he "bears No Fault or Negligence for the violation" and can establish how the Prohibited Substance entered his system, the period of Ineligibility can be eliminated. "No Fault or Negligence" means: The Player's establishing that he did not know or suspect, and could not reasonably have known or suspected even with the exercise of utmost caution, that he had used or been administered the Prohibited Substance. 36. The second category is where the Player "bears No Significant Fault or Negligence" in which case the period of Ineligibility may be reduced, but the reduced period of Ineligibility may not be less than one-half of the minimum period of Ineligibility otherwise applicable. The definition of the term provides: The Player's establishing that his fault or negligence, when viewed in the totality of the circumstances and taking into account the criteria for No Fault or Negligence, was not significant in relationship to an antidoping rule violation. 37. A footnote to the corresponding provision of the WADA Code makes it clear that only in truly exceptional cases and not in the vast majority of cases will these provisions operate to eliminate or reduce a sanction. An example of where the elimination of a sanction might be justified would be where a Player was sabotaged by a competitor. The administration of a Prohibited Substance by an athlete's personal physician without and aldara.
Potassium supplements potassium-sparing diuretics, such as spironolactone aldactone ® , triamterene dyrenium ® , and amiloride midamor ® , among others lithium eskalith ® , lithane ® , lithonate ® , lithotabs ®.
I thank Florian Pittet for the photo. I declare that I have no conflict of interest. 1 2 3 Black R, Morris S, Bryce J. Where and why are 10 million children dying every year? Lancet 2003; 361: 222634. Luby SP, Agboatwalla M, Feikin DR, et al. Effect of handwashing on child health: a randomised controlled trial. Lancet 2005; 366: 22533. Luby SP, Agboatwalla M, Painter J, Altaf A, Billhimer WL, Hoekstra RM. Effect of intensive handwashing promotion on childhood diarrhea in high-risk communities in Pakistan: a randomized controlled trial. JAMA 2005; 291: 254754.
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Australia -- A recent letter to the Medical Journal of Australia has highlighted a case of peripheral neuropathy in association with nitrofurantoin, an antibiotic used for urinary tract infection prophylaxis 1 ; . This is a well known effect of nitrofurantoin but awareness may be declining due to reduced use. The Australian Adverse Drug Reactions Committee ADRAC ; has received 18 reports of peripheral neuropathy since 1978. While there were no reports received between 1990 and 1997, there have been three in the last 4 years. Most of the reports have involved elderly females. Daily dosages have ranged from 100 mg to 400 mg with a median of 250 mg. The time to onset has ranged from 3.
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Development of a whole-virus multiplex flow cytometric assay for antibody screening of a specific pathogen-free primate colony Kuller L., Watanabe R., Anderson D., Grant R.; Diagn. Microbiol. Infect. Dis. 53 3 185-193 ; , 2005 [L. Kuller, Washington National Primate Research Center, University of Washington, Box 357331, Seattle, WA 98195, United States] Weitzenburger D., Vits A., Hamann H., Distl O.; Berl. Munch. Tierarztl. Wochenschr. 118 11-12 441-448 ; , 2005 [Dr. O. Distl, Institut f r Tierzucht und Vererbungsforschung, Stiftung u Tier rztliche Hochschule Hannover, B nteweg 17p, 30559 a u Hannover, Germany] Solis- Calderon J.J., Segura- Correa V.M., Segura- Correa J.C.; Prev. Vet. Med. 72 3-4 253-262 ; , 2005 [J.C. Segura- Correa, Facultad de Medicina Veterinaria Y Zootecnia, Universidad Autonoma de Yucatan Zootecnia, UADY, Km. 15.5 carretera Merida- Xmatkuil, M rida, Yucat n, Mexico] e a Weber S.L., Bryant B.J., Indrikovs A.J.; Transfusion 45 8 1327-1330 ; , 2005 [Dr. B.J. Bryant, MT ASCP ; SBB, University of Texas Medical Branch, 301 University Boulevard, Galveston, TX 77555- 0609, United States] Moen L.H., Sletten G.B., Miller I., et al.; Food Agric. Immunol. 16 2 83-90 ; , 2005 [L.H. Moen, National Veterinary Institute, PO Box 8156 Dep., N- 0033 Oslo, Norway] Cliquet P., Goddeeris B.M., Bonroy K., Cox E.; Food Agric. Immunol. 16 2 101-115 ; , 2005 [E. Cox, Laboratory of Veterinary Immunology, Faculty of Veterinary Medicine, Ghent University, Salisburylaan 133, B- 9820 Merelbeke, Belgium] De Meulenaer B., De La Court M., Acke D., et al.; Food Agric. Immunol. 16 2 129-148 ; , 2005 [B. De Meulenaer, Laboratory of Food Chemistry, Department of Food Safety and Food Quality, Ghent University, Coupure Links 653, B- 9000 Gent, Belgium] Lee N.A., Rachaputi N.C., Wright G.C., et al.; Food Agric. Immunol. 16 2 149-163 ; , 2005 [N.A. Lee, University of New South Wales, School of Chemical Engineering and Industrial Chemistry, Sydney, NSW 2052, Australia] Krause K., Marcu K.B., Greeve J.; Mol. Immunol. 43 4 295-307 ; , 2006 [J. Greeve, Department of Clinical Research, University of Berne, Berne, Switzerland] 2986.
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12. Shire U.S., Inc. v. Barr Laboratories, Inc., No. 02-2023, slip op. at 1011 D. N.J. Sept. 16, 2003 ; . 13. Id. at 13. 14. Id. at 910. 15. Id. at 1112. 16. Id. 17. Ives Laboratories, Inc. v. Darby Drug Co., 488 F.Supp. 394 E.D.N.Y. 1980 ; . 18. Id. at 39899. 19. Shire U.S., Inc. v. Barr Laboratories, Inc., No. 02-3647, slip op. at 1617 3d Cir. May 23, 2003 ; . 20. Drugs in Schedule II of the Controlled Substances Act are those substances that have a high potential for abuse with severe liability to cause psychic or physical dependence, but have some approved medical use.
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ENURESIS PREVALENCE AND ACCOMPANYING FACTORS IN SCHOOLCHILDREN: A QUESTIONNAIRE STUDY FROM SOUTHEAST ANATOLIA K.U. zkana, M. Garipardicb, A. Toktamisd, H. Karabiberb, T. Sahinkanatc Departments of aPediatric Surgery, bPediatrics, and cUrology, Faculty of Medicine, Kahramanmara St Imam University, Kahramanmara , and dDepartment of Family Physician, Medical Faculty, Cumhuriyet University, Sivas, Turkey Urologia Internationalis 2004; 73: 149-155 Introduction: The aims of this study were to establish the prevalence of enuresis, to determine accompanying factors, and to identify common methods in the management of enuresis in Turkish children aged between 6 and 11 years living in eastern Anatolia. Methods: The parents of 3, 527 schoolchildren aged between 6 and 11 years completed a self-administered questionnaire. The questionnaire asked about sociodemographic data, enuresis data, physical or psychological disorders, and family stressors. Descriptive statistics and 2 test were used for data analysis. Results: The response rate was 88%. The overall prevalence of reported enuresis was 12.96%, and the prevalence of marked enuresis at least weekly ; was 9.8%. Enuresis was notably more common in boys male: female ratio 1.6 ; , and the prevalence rates decreased by age without gender bias. Of all enuretic children, 21% had also daytime bed-wetting. The rate of a positive family history was 42% for siblings only and 66% for the other family members. Significantly more of the dry children woke up spontaneously at night to void as compared with the enuretic children p 0.001 ; . The parental concern level was not high, and only 15% of the children visited a physician for the management of enuresis. Low socioeconomic status, unfavorable perinatal or postnatal history, and unsatisfactory familial characteristics were significantly more frequent in the enuretic group p 0.05 ; . The enuretic children had also higher rates of poor school performance and poor social adaptation as compared with nonenuretic children p 0.001 ; . Conclusions: These results suggest that the prevalence of enuresis in eastern Anatolia is similar to that reported from western Anatolia and from most other countries. Turkish families did not report a high-level concern about enuresis, and the problem was managed primarily within the family.
1. Chou LM. A Guide to the Dangerous Marine Animals of Singapore. Singapore: Singapore Science Centre, 1993. 2. Brentjens M, Sra KK, Junior VH, Lee P, Tyring SK. Marine freshwater dermatology. In: Tyring SK, Lupi O, Hengge UR, eds. Tropical Dermatology. Philadelphia: Elsevier, 2006: 455-67. 3. Scharf MJ. Cutaneous injuries and envenomations from fish, sharks and rays. Dermatol Ther 2002; 15: 47-57. Halstead BW, Vinci JM. Venomous fish stings ichthyoacanthotoxicoses ; . Clin Dermatol 1987; 5: 29-35. Auerbach PS. Marine envenomations. N Eng J Med 1991; 325: 486-93. Burke WA. Cnidarians and human skin. Dermatol Ther 2002; 15: 18-25. Fisher AA. Aquatic dermatitis, Part 1. Dermatitis caused by coelenterates. Cutis 1999; 64: 84-6. Fenner PJ, Williamson JA. Worldwide deaths and severe envenomation from jellyfish stings. Med J Aust 1996; 165: 658-61. Hartwick R, Callanan V, Williamson J. Disarming the box-jellyfish: nematocyst inhibition in Chironex fleckeri. Med J Aust 1980; 1: 15-20. Nomura JT, Sato RL, Ahern RM, et al. A randomized paired comparison trial of cutaneous treatments for acute jellyfish Carybdea alata ; stings. J Emerg Med 2002; 20: 624-6. Auerbach PS. A Medical Guide to Hazardous Marine Life. 3rd ed. Arizona: Best Publishing, 1997. 12. Reed KC, Crowell MC, Castro MD, Sloan ML. Skin and soft-tissue infections after injury in the ocean: culture methods and antibiotic therapy for marine bacteria. Mil Med 1999; 164: 198-201.
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