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If you experience any of the following serious side effects, stop taking carbidopa, entacapone, and levodopa and seek emergency medical attention or contact your doctor immediately: an allergic reaction difficulty breathing; closing of the throat; swelling of the lips, tongue, or face; or hives uncontrolled movements of a part of the body; fever; seizures; severe nausea, vomiting, or diarrhea; an irregular heartbeat or fluttering in the chest; hallucinations; unusual changes in mood or behavior; or depression or suicidal thoughts.
Ann O'Hanlon & Peter Coleman University of Southampton, UK & Royal College of Surgeons in Ireland, Dublin, Ireland Objectives: This cross-cultural study sought to examine adults' attitudes towards their own ageing and future old age in the UK and the US. This research also sought to examine the role of specific concerns across physical, social & psychological domains ; in contributing to general evaluations about own future old age. Method: Adults of all ages n 325 ; were recruited in the same way from high streets in two cities in the US Washington, D. C. and New York ; and the UK Southampton and Sheffield ; . A wide range of measures were used, including scales specifically developed from earlier exploratory data. Results: Across both cultures, results indicate 1 ; that the new scales did have good psychometric properties, 2 ; that perceived problems across physical, social & psychological domains contributed similarly to general attitudes about own ageing and future old age, and 3 ; that concerns about own future old age better predicted general attitudes than did evaluations about possible positive experiences in later life. Results also indicated that the attitudes of participants in both countries were often similar, although the US respondents had some more negative attitudes than their UK-based counterparts. Conclusion: To bring quality of life and health to increased longevity, more research is needed exploring attitudes to own old age. The role of society and culture needs to be considered in such research. The current presentation offers new measures and insights which can facilitate further work in this area.
Bond maturing in 2008 started the year at a price of roughly 104 cents in the euro; by the end of March it had risen to 106, although it is now trading back at around 104. Maxwell joined Deutsche Bank in 2000 from BNP Paribas, where he spent three years as a senior analyst covering industrial credits. Before that, he spent three years at Equitable Life as a credit analyst and portfolio manager. His early banking career was spent managing distressed credits at Hill Samuel Merchant Bank. His work at Deutsche Bank focuses on relative value, supported by fundamental analysis, for example, carbidopa generic.
Recent therapeutic guidelines based on cr first data recommend using sustained-release carbidopa-levodopa, although this is more expensive 2.
The effectiveness of levodopa administered with carbidopa and entacapone in the treatment of parkinson’ s disease was established in three 24-week multicenter, randomized, double-blind placebo-controlled trials in patients with parkinson’ s disease experiencing wearing off and levodopa.
Table I. JH and pH during Withdrawal of Glybenclamide.
One more ancient drug that today we judge to be too dangerous for general use, described by the chinese nearly 5000 years ago, is marijuana also known as hashish, ganja, or kif ; made from the resinous oil of the cannabis plant and carvedilol, for example, carbidopa levodopa.
Calcium Gluceptate Calcium Lactobionate Calcium Pantothenate Calcium Saccharate Candicidin Cannabidiol Controlled Substance CI Authentic Substance. For Qualitative Use Only ; Cannabinol Controlled Substance CI Authentic Substance ; Capreomycin Sulfate Capsaicin Captopril Captopril Disulfide Limit test Carbachol Carbamazepine Carbarsone Carbenicillin Indanyl Sodium Carbenicillin Monosodium Monohydrate Csrbidopa Carbinoxamine Maleate Carboplatin Carboprost Tromethamine Carisoprodol Carphenazine Maleate Carteolol HCl Cathinone HCl Controlled Substance CI Limit test Formerly Cat. No. 02620-8 ; Cefaclor Cefaclor, Delta-3 Isomer Cefadroxil Cefamandole Lithium Cefamandole Nafate Cefamandole Sodium For Identification Use Only ; Cefazolin Cefoperazone Dihydrate Cefonicid Sodium Cefmenoxime HCl Cefmetazole Ceforanide Cefotaxime Sodium Cefotetan Cefotiam HCl Cefprozil E-isomer Cefprozil Z-isomer Cefoxitin Ceftazidime, Delta-3-Isomer Ceftazidime Pentahydrate Ceftizoxime Ceftriaxone Sodium E-Isomer For System Suitability Use Only ; Cefuroxime Sodium Cefuroxime Axetil Cefuroxime Axetil Delta-3-Isomers Cellulose Acetate Cellulose Acetate Phthalate Cephaeline Hydrobromide Limit test Cephalexin Cephalothin Sodium Cephapirin Benzathine Cephapirin Sodium Cephradine Cetyl Alcohol Cetylpyridinium Chloride Chlorambucil.
2 inhibiting the metabolism of levodopa to 3-OMD through the COMT pathway. This leads to more sustained plasma concentrations of levodpa. It is believed that at a given frequency of levodopa administration, these more sustained plasma levels of levodopa result in more constant dopaminergic stimulation in the brain leading to greater effects on the signs and symptoms of Parkinson's Disease. The higher levodopa levels also lead to increased levodopa adverse effects, sometimes requiring a decrease in the dose of levodopa. In animals, while entacapone enters the CNS to a minimal extent, it has been shown to inhibit central COMT activity. In humans, entacapone inhibits the COMT enzyme in peripheral tissues. The effects of entacapone on central COMT activity in humans have not been studied. PHARMACODYNAMICS Effect of Entacapone on erythrocyte COMT activity Studies in healthy volunteers and patients with Parkinson's disease have shown that entacapone dose-dependently and reversibly inhibits human erythrocyte COMT activity after oral administration. Following single doses of 200 and 800 mg of entacapone, maximal inhibition of erythrocyte COMT activity was 64% and 82%, respectively. Effect of entacapone on the pharmacokinetics of levodopa and its metabolites When 200 mg entacapone is administered together with levodopa carbidopa, it increases the area under the curve AUC ; of levodopa by approximately 35% and the elimination half-life of levodopa is prolonged from 1.3 h to 2.4 h. In general, the average peak levodopa plasma concentration and the time of its occurrence Tmax of 1 hour ; are unaffected. The onset of effect occurs after the first administration and is maintained during long-term treatment. In a dose-response study in patients with Parkinson's disease, the maximal effect was obtained with a single dose of 200 mg entacapone. Doses of entacapone greater than 200 mg did not further improve the bioavailability of levodopa and cilostazol.
Carbidopa levodopa carbidopa levodopa skip to: introduction interactions summary vitamin interactions food interactions references also indexed as: apo-levocarb, atamet, co-careldopa, endo levodopa carbidopa, half sinemet, nu-levocarb, sinemet see also: carbidopa skip to: introduction interactions summary vitamin interactions food interactions references levodopa is required by the brain to produce dopamine, an important neurotransmitter!
Was the SAE one of the recognised undesirable effects of the trial medication? and ciprofloxacin.
Pharmacokinetic behaviour does not change after repeated administration.
Agent Levodopa preparations Carbidops levodopa Sinemet Sinemet CR Dopamine agonists Bromocriptine Pergolide Permax ; Pramipexole Mirapex ; Ropinirole Requip ; Anticholinergics Trihexyphenadyl Benztropine Amantadine 2 mg 0.5 mg 100 mg 15.39 7.00 16.88 mg 0.25 mg 1 mg 1 mg 5 mg 168.00 120.00 370.00 mg 100 mg 25 mg 100 mg 50 mg 200 mg 27.54 74.00 158.00 Dose Cost and clarinex.
Typical medications for treating myoclonus activate inhibitory neuronal networks. Two of the most effective compounds are sodium valproate and benzodiazepines. These medications are thought to reduce myoclonus because they activate GABAnergic systems, but it has been suggested that the antimyoclonic action of drugs such as clonazepam might be mediated by enhancement of serotonergic rather than GABAergic neurotransmission. Other antiepileptic drugs, including phenobarbital, piracetam, levetiracetam, phenytoin, carbamazepine, and zonisamide, have also been effective in people with myoclonus.1 With the exception of visual cortical reflex myoclonus, 2 dopaminergic agents such as carbidopa levodopa are not thought to be therapeutic in the treatment of myoclonus and even have been reported to induce myoclonus in some patients with Parkinson's disease.3 Recently, we evaluated a woman whose myo.
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The authors acknowledge the Netherlands Ministry of Health for their financial support for this project. P, .S469 and clindamycin.
Sinemet caused both visceral and skeletal malformations in rabbits at all doses and ratios of carbidopa levodopa tested, which ranged from 10 times 5 times the maximum recommended human dose of carbidopa levodopa to 20 times 10 times the maximum recommended human dose of carbidopa levodopa.
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By helping more levodopa get into the brain so that less stays in the bloodstream, carbidopa can reduce some of levodopa's side effects such as nausea and clobetasol.
Tions. Examination of the one Level A recommendation reveals additional questions about the knowledge base on which this recommendation is based.5 This Level A recommendation is that the COMT inhibitor entacapone and the MAO-B inhibitor rasagiline be offered to reduce "off" time. The same Parameter includes a Level C recommendation that sustained release carbidopa levodopa may be disregarded as a treatment to reduce "off" time. The idea that extended release carbidopa levodopa is ineffective in reducing "off" time flies in the face of considerable clinical experience. The different levels of the recommendations are a function of the available clinical trial data. The entacapone and rasagiline recommendations are based on the existence of replicated successful Class I trials for these agents. There have been no Class I or II trials of the efficacy of extended release carbidopa levodopa in reducing "off" time. This evidentiary discrepancy probably reflects the fact that entacapone and rasagiline are novel entities, not adaptations of an existing agent. As recently developed agents, they faced greater scrutiny from regulators and from the movement disorders community than did extended release carbidopa levodopa. In addition, we do not know the best approach for reducing "off" time. This review panel was unable to issue a strong recommendation because of inadequate comparative data. The lack of adequate data for formulating strong recommendations is not unique to the treatment of PD. Does this mean that these, and by implication other, Practice Parameters are fruitless? Absolutely.
As you probably know, "levodopa" refers to a family of drugs whose common ingredient includes carbidopa and levodopa. Brand-named Sinemet, it is the oldest and strongest medication for Parkinson disease. The drug may come in the traditional yellow immediate release form. Or it may be the controlled release formulation referred to as "CR" or "ER" ; , usually in a lavender or cream color. It may be augmented with entacapone, as it is in Stalevo, to extend its period of action. Or it can take a "dissolve-on-the-tongue" form as it does with Parcopa. All these formulations represent "levodopa therapy." Synthetic agonists, on the other hand, include Mirapex, Requip and Permax, as well as a newly anticipated "patch" formulation called Rotigotine. Apomorphine is another synthetic that is injectable. An "agonist" is a name given to a drug that acts like, or in place of, another drug. All these medications represent chemical imitations of dopamine and mimic its action to some degree. Since they all imitate dopamine but are not, chemically speaking, true dopamine, they bear the name "synthetic and clotrimazole.
This equation should be valid at least for scales bigger than the Planck scale: Since general relativity is the established classical theory of gravity on large scales, and since we assume quantum theory to be universally valid, the direct quantization of general relativity should be valid at least as an effective theory on large scales. It may, however, break down near the Planck scale. An ad hoc modification can be made, for example, by the introduction of an appropriate `Planck potential' in order to facilitate the normalizability of the wave function there [9]. A more fundamental approach would be to employ results from the full theory before the restriction to homogeneous models. This can be achieved in loop quantum gravity where the spectra of geometrical operators turn out to be discrete, cf. [3, 10] and the references therein. For cosmological models one then finds that the WheelerDeWitt equation 2 ; is replaced by a difference equation for steps characterized by n Z [10]. The number n is related to the eigenvalue of the operator p, 1 2 p|n lP n|n , 6 3.
Calan . 19, 20 Calan IV . 19, 20 CALAN SR. 20 Calcijex . 25 Calcijex IV . 25 calcitriol. 25 CALCITRIOL. 25 CAMPRAL . 23 CAMPTOSAR . 13 CANASA . 28 CAPITROL . 22 CARAC. 12 Carafate . 24 carbachol . 30 carbamazepine . 9 CARBATROL . 9 carbidopa levodopa . 14 carboplatin. 13 Cardizem . 19, 20 CARDIZEM CD . 19, 20 Cardizem IV. 19, 20 CARDIZEM LA . 19 Cardura. 16, 19, 24 carisoprodol. 32 Carmol 40. 30 CASODEX. 26 Catapres. 17, 18 Ceclor. 8 cefaclor . 8 cefadroxil hydrate . 8 cefotaxime sodium. 8 cefpodoxime proxetil. 8 Ceftin. 8 cefuroxime axetil. 8 CELEBREX. 7, 11 Celexa . 10 CELLCEPT. 27 CELONTIN. 9 cephalexin monohydrate . 8 Cephulac . 24 CEREZYME . 23 chloral hydrate. 32 chlorhexidine gluconate. 22 chloroquine phosphate. 14 cholestyramine sucrose. 20 ciclopirox . 22 and cutivate and carbidopa.
Featuring Keynote Speaker Dr. David Lipschitz, Director of the Center on Aging at the University of Arkansas for Medical Sciences & Author.
Table 6.3: Risk of stroke and bleeding in meta-analysis of primary and secondary prevention trials and cyproheptadine.
Dr. Kuban l ; and Dr. Pettaway r ; review bone scans with Dr. Christopher Stage center ; , a resident in the Department of Urology. approach does not allow lymph node sampling. But in patients with low-risk disease this is acceptable. Laparoscopic surgery is increasingly being used for radical prostatectomy. "With laparoscopy, you can sample the lymph nodes and remove the prostate as completely as you would with an open incision, using five smaller incisions, " Dr. Pettaway said. "Laparoscopic prostatectomy is a challenging procedure and requires specialized training." Early data seem to show that there is less blood loss with this procedure than with conventional surgery. It also appears that the laparoscopic procedure allows a quicker recovery. "My feeling in talking to patients is that the ones who undergo laparoscopic prostatectomy may be getting back to normal activity a little bit quicker, maybe a week or two sooner than with the open approach, " Dr. Pettaway said. Cryosurgery is making a resurgence due to technological advances. Previously, it was impossible to be certain the entire prostate gland was being frozen without also destroying surrounding structures. As in the external beam radiation therapy, the advent of sonography has allowed for much more precise monitoring of the procedure. In addition, the development of a urethral warmer and temperature probe monitors placed near the rectum and external sphincter have helped decrease complications. The next step into the future of prostate surgery is robotic surgery. "The surgeon actually performs the surgery by sitting at a remote console, " Dr. Pettaway said. "The robot is at the bedside with a human assistant." Ports are placed just as in laparoscopic surgery, and the robot's hands manipulate the instruments in response to movements made by the surgeon at the remote console. The biggest benefit is that the surgeon is looking at a threedimensional image rather than trying to interpret a two-dimensional image, as in laparoscopy. "Most urologists who have tried robotic surgery say they really like it, " Dr. Pettaway said. Nerve grafts, gene therapy, and other advances One of the biggest concerns among patients being treated for prostate.
Even in these patients, a reduction of carbidopa-levodopa or entacapone may be necessary however, the provider is reminded that this may not be possible with stalevo.
How should i take carbidopa, entacapone, and levodopa.
Stages V or VI occur in only a minority of cases Figure 1 ; . Alzheimer pathology is not a prerequisite for the existence of dementia however, since cases with "pure" LB disease may present clinically with cognitive impairment and other neuropsychiatric features. Nor is the number of cortical LBs robustly correlated with either the severity or the duration of dementia, 13, 14 although associations have been reported with LB and plaque density in midfrontal cortex.15 LN and neurotransmitter deficits are suggested as more likely correlates of clinical symptoms.14, 16 -Synuclein immunoreactive deposits with many of the characteristics of LBs have also been reported in a high proportion of AD cases, particularly in the amygdala.17 In this context, they may represent an end-stage phenomenon, with secondary accumulation of aggregated synuclein in severely dysfunctional neurones that are already heavily burdened by plaque and tangle pathology.18 Whatever the explanations are for this considerable overlap in pathological lesions in DLB and AD, it is clear that clinical separation of cases is going to be less than 100% precise. The presence of Alzheimer pathology in DLB appears to modify the typical clinical presentation making such cases harder to differentiate clinically, 19 with the core features see below ; being scant or absent and the clinical picture more closely resembling AD. DLB and Parkinson's disease dementia The clinical and pathological classification of DLB is further complicated by its relationship with idiopathic Parkinson's disease, a disorder in which dementia may develop in up to 78% patients20 and which is similar to DLB21, 22 in respect of fluctuating neuropsychological function, 23 neuropsychiatric features, 24 and extrapyramidal motor features Table I ; .25 There is considerable debate as to the relationship between the two diagnoses.26 An arbitrary "1-year rule" is frequently used to "separate" them by proposing that onset of dementia within 12 months of parkinsonism qualifies as DLB and more than 12 months of parkinsonism before dementia qualifies as Parkinson's disease dementia PDD ; . This is certainly helpful in individual clinical case diagnosis and management, but is increasingly hard to justify from a neurobiological point of view. There do, because carrbidopa 25 mg.
Calcitriol Captopril Captopril HCTZ Carbamazepine Ccarbidopa levodopa Carboptic Carisoprodol Carisoprodol aspirin Cefaclor Cefadroxil Cefuroxime Cephalexin Cesia Chloral hydrate Chlordiazepoxide Chlordiazepoxide clidinium Chloroquine Chlorothiazide Chlorphen phenyleph methscop Chlorpromazine Chlorpropamide Chlorthalidone Cholestyramine Choline & magnesium Citalopram Citrate citric acid Clarithromycin, XL Clemastine 2.68mg Clindamycin Clobetasol Clomipramine Clonazepam Clonidine Clorazepate SD Tier Three ; Clozapine Codeine Colchicine and levodopa.
Verify: a signature h, S ; on a message M is accepted iff h H2 M, e S, Qpub ; g -h ; . The scheme can be thought of as an identity-based extension of a digital signature discussed in two independent papers [9, 38]. More precisely, the method for obtaining private keys from identities is a simplification of a method suggested by Sakai and Kasahara [33] ; . In [25], Kurosawa and Heng described an identity-based identification IBI ; protocol that implicitly suggests an IBS described in appendix E and which can be proven secure under the same assumption as our proposal. It turns out that ours is slightly faster than the Kurosawa-Heng IBS in the signature generation. At Eurocrypt'04, Bellare, Namprempre and Neven established a framework [5] for proving the security of a large family of identity-based signatures and they only found two schemes to which their framework does not apply. The present one does not either fall into the category of schemes to which it applies. Indeed, it can be showed that our IBS does not result from the transformation of any convertible standard identification or signature scheme in the sense of [5] ; unless the q-SDHP is easy. A direct security proof is thus needed. 3.1 Security results.
EPM1400000 Methylatropine nitrate EPM1450000 Methylcarbidopa EPM1470000 Methyl 2- 4-chlorophenoxy ; -2methylpropionate EPM1490000 Methyl 3, EPM1500000 EPM1550000 EPM1551000 EPM1600000 Methyldopa Methylene chloride 2- 1-Methylethyl ; pentanoic acid 20 Methylnaphthalene, n D: 1.6165.
Data reported by the projects indicate that juveniles have a higher rate of successfully completing their program than do adults. Adults are reported to have stayed in their program a greater number of days before being unsuccessfully terminated or absconding than do juveniles. Ohio Alternatives to Detention, Jail and Prison Reason for Unsuccessful Termination CY 2003 Reason for Unsuccessful Termination Lack of cooperation Unexcused absences Failed urinalysis Other indication of drug use * Percents do not total 100 due to rounding. Juveniles 79% 0% 7% 14% Adults 5% 29% 2% For those offenders unsuccessfully terminated from their program, the most common reason for termination of juvenile offenders was lack of cooperation while for adult offenders the most common reason was for indication of drug use other than urinalysis Summary of Evaluations Evaluation of Cognitive-Behavioral Treatment Programs University of Cincinnati There are interventions that have shown positive effects in the correctional program evaluation. One such treatment is cognitive-behavioral intervention. Although this treatment has been found to have positive effects, the extent of these effects is unknown. Moreover, which types of offenders this treatment works best for is also unknown. The University of Cincinnati's Division of Criminal Justice received Byrne funding to study the applicability of cognitive-behavioral programs in a variety of treatment settings with many offender populations. During CY 2003 data was collected on offenders from five sites using the cognitive-behavioral approach and a comparison group of offenders who did not receive cognitive-behavioral programming. After the data collection phase is complete, analysis will focus on two primary questions. Does the cognitivebehavioral group have lower recidivism rates? Does the cognitive-behavioral treatment work best with particular types of offenders? 17.
CALCITRIOL CAL-NATE CAMILA CAMPTOSAR CANASA CAPTOPRIL CAPTOPRIL HYDROCHLOROTHIAZIDE CARBAMAZEPINE CARBATROL CARBIDOPA LEVODOPA CARBOCAINE CARBOPLATIN CARIMUNE NF NANOFILTERED * CARIMUNE * CARISOPRODOL COMPOUND CODEINE CARISOPRODOL * CARTEOLOL HCL CASODEX CEENU CEFACLOR CEFACLOR CEFACLOR ER CEFADROXIL CEFADROXIL MONOHYDRATE CEFPODOXIME PROXETIL CEFUROXIME CELEBREX * CELLCEPT CELLCEPT CELLCEPT CELONTIN CEPHALEXIN CEPHALEXIN CEPHRADINE CEREBYX CEREDASE CEREZYME CHLORAMPHENICOL SOD SUCCINATE CHLORHEXIDINE DIGLUCONATE CHLORHEXIDINE GLUCONATE CHLOROPROCAINE HCL CHLOROQUINE PHOSPHATE CHLOROTHIAZIDE CHLORPROMAZINE HCL CHLORTHALIDONE CHLORZOXAZONE * CHOLESTYRAMINE CHOLESTYRAMINE LIGHT CHOLINE MAG TRISALICYLATE CICLOPIROX CAPSULE TABLET TABLET VIAL SUPP.RECT TABLET TABLET TAB CHEW CAP.SR 12H TABLET SA VIAL VIAL VIAL VIAL TABLET TABLET DROPS TABLET CAPSULE CAPSULE SUSP RECON TAB.SR 12H CAPSULE TABLET TABLET TABLET CAPSULE CAPSULE TABLET SUSP RECON CAPSULE TABLET SUSP RECON CAPSULE VIAL VIAL VIAL VIAL SOLUTION LIQUID VIAL TABLET TABLET TABLET TABLET TABLET PACKET PACKET TABLET CREAM GM.
COMT catechol-O-methyltransferase; PDI peripheral decarboxylase inhibitor e.g., carbixopa and benserazide.
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Parallel Parking BT: On Street Parking Paralysis Paramedical Personnel Paramedics Paramilitary Forces SN: Distinct from the regular armed forces of any country but resembling them in organization, equipment, training, or mission. USE: Military Organizations Paranoid Behavior BT: Behavioral Symptoms Paranoid Personality Disorder BT: Personality Disorders Paraphilias BT: NT.
Gastrointestinal injury, ulcers and related bleeding are serious health matters that often strike patients by surprise, including those with or without any past symptoms or underlying digestive condition. The two primary causes of ulcers are damage from 1 ; the presence of a microorganism known as Helicobacter pylori; or 2 ; from regular use of non-steroidal anti-inflammatory drugs known as NSAIDS ; , including aspirin. While some damage may occur with modest, short-term doses, problems are more likely to arise in regular NSAID users, and increase with the magnitude of use--more frequent use and or higher dosages. NSAIDs and aspirin have some very positive health benefits. Like all medications, care must be taken with their use. They should not be taken with alcohol, as the combination can increase the risk of GI bleeding. Patients who need to use NSAIDs regularly should consult with their physician on a regular basis to be alert for any potential GI effects. Problems may arise with few, if any, symptoms, but if they are recognized early, there are a variety of ways to minimize or reverse any adverse effects. Options include using alternatives to NSAIDs, or your physician prescribing medications that can reduce any adverse effects.
Carr 229 230 231 TPL Name PRESCRIPTION HEALTH SERVICES PYRAMID LIFE INSURANCE COMPANY SAVERS LIFE INSURANCE COMPANY GENERAL ADJUSTMENT BUREAU STERLING LIFE INS. CO.HEALTH PLAN OPERATIONS PRONET PPO ; SHAW INDUSTRIES GUARANTEE TRUST LIFE INSURANCE Address Line PO BOX 80716 PO BOX 772 8064 NORTH POINT BLVD SUITE 201 PO BOX 81808 PO BOX 5348 PO BOX 101387 PO BOX 10 1275 MILWAUKEE AVENUE PO BOX 8019 PO BOX 8019 PO BOX 1028 1 HORACE MANN PLAZA PO BOX 55270 PO BOX 792070 8228 MAYFIELD RD. STE 5B AMERICAN GENERAL CENTER PO BOX 10844 PO BOX 23070 PO BOX 23070 PO BOX 11010 5050 SPRING VALLEY ROAD 25145 COUNTRY CLUB BLVD 1333 S. KIRKWOOD ROAD PO BOX 12922 City LOS ANGELES SHAWNEE MISSION WINSTON SALEM ALTANTA BELLINGHAM FORT WORTH DALTON GLENVIEW APPLETON APPLETON WEST TRENTON SPRINGFIELD PHOENIX SAN ANTONIO CHESTERLANDE NASHVILLE CLEARWATER TUCSON TUCSON TUCSON DALLAS NORTH OLMSTED ST. LOUIS PENSACOLA State CA KS NC 54913 08628 Zip 90080 66201 27106 CODE IN OPEN STATUS BY SCHA 6157491000 8776045240 CODE NOT REQUESTED BY MEDICAID. ASSIGNED BY SCHA 8005544954 8008449636 CARRIER WAS FIRST INTEGRATED HEALTH 8002558063 DORMANT 8 06 8006581413 CODE IN OPEN STATUS BY SCHA MEDICARE SUPPLEMENTAL PLAN G 8006399157 CODE ASSIGNED BY SCHA 8772926037 Phone Num Carrier Comment 8004212342 CODE ASSIGNED BY SCHA 8004440321 8006420483 4044579555 CODE ASSIGNED BY SCHA CODE NOT REQUESTED BY MEDICAID. ASSIGNED BY SCHA.
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