DRAFT FOR SECOND CONSULTATION 1 2 3 LARC: Full guideline DRAFT May 2005 ; Diabetes 6.11 Medical conditions and contraindications Recommendation: Breastfeeding women may be advised that they can use injectable contraceptives imediately after childbirth if other methods are unacceptable. [D GPP].
KREMERS URBAN DOAK DERM. DOAK DERM. BIOGLAN PHARM PRESCRIPT SOLN PRESCRIPT SOLN PRESCRIPT SOLN PRESCRIPT SOLN PRESCRIPT SOLN PRESCRIPT SOLN PRESCRIPT SOLN PRESCRIPT SOLN PRESCRIPT SOLN PRESCRIPT SOLN PRESCRIPT SOLN PRESCRIPT SOLN PRESCRIPT SOLN PRESCRIPT SOLN PRESCRIPT SOLN PRESCRIPT SOLN PRESCRIPT SOLN ANI PHARMACEUTI ANI PHARMACEUTI AHP AHP AHP AHP AHP AHP AHP AHP AHP AHP AHP AHP AHP AHP AHP AHP AHP AHP AHP AHP AHP AHP AHP AHP AHP AHP AHP AHP AHP AHP AHP AHP AHP AHP AHP, for example, pravachol weight gain.
1. 2. 3. McKusick V. Mendelian Inheritance in Man, 6th Ed. Baltimore: Johns Hopkins University Press, 1983; 506-7. Rendu HJLM. Epistaxis rpts chez un sujet porteur de petits angiomes cutans et muqueux. Bull Soc Med Hop 1896; 13: 731-3. Haitjema T, Westermann CJ, Overtoom TT, et al. Hereditary hemorrhagic telangiectasia Osler-Weber-Rendu disease ; : New insights in pathogenesis, complications, treatment. Arch Intern Med 1996; 156: 714-9. Peery W. Clinical spectrum of hereditary hemorrhagic telangiectasia Osler-Weber-Rendu disease ; . J Med 1987; 82: 989-97. Haitjema T, Disch E, Overtoom TT, Westermann CJ, Lammers JWJ. Screening of family members of patients with hereditar y hemorrhagic telangiectasia. J Med 1995; 99: 519-24. Reilly PJ, Nostrant TT. Clinical manifestations of hereditary hemorrhagic telangiectasia. Gastroenterol 1984; 79: 363-7. Wang HC, Yang PC, Kuo SH, Luh KT. Pulmonary arteriovenous malformation: Analysis of 10 cases. J Formos Med Assoc 1998; 97: 97100. Shar ma VK, Howden CW. Gastrointestinal and he patic manifestations of hereditary hemorrhagic telangiectasia. Dig Dis 1998; 16: 169-74. McDonald MT, Popenberg KA, Glosk S, et al. A disease locus for hereditary hemorrhagic telangiectasia maps to chromosome 9q3334. Nat Genet 1994; 197-202. Porteous ME, Curtis A, Williams O, Marchuk D, Bhattacharya SS, Burn J. Genetic heterogeneity in hereditar y hemorrhagic telangiectasia. J Med Genet 1994; 31: 925-6. McAllister KA, Lennon F, Bowles-Biesecker B, et al. Genetic heterogeneity in hereditary hemorrhagic telangiectasia: Possible correlation with clinical phenotype. J Med Genet 1994; 31: 927-32. Heutink P, Haitjema T, Breedveld G, et al. Linkage of hereditary hemorrhagic telangiectasia to chromosome 9q34 and evidence for locus heterogeneity. J Med Genet 1994; 31: 933-6. Cymerman U, Vera S, Pece-Barbara N, et al. Identification of hereditary hemorrhagic telangiectasia type 1 in newborns by protein expression and mutation analysis of endoglin. Pediatr Res 2000; 47: 2435. Hodgson CH, Kaye RL. Pulmonary arteriovenous fistula and hereditary hemorrhagic telangiectasia: A review and report of 35 cases of fistula. Dis Chest 1963; 43: 449-55. Wyngaarden J, Lloyd L, Bennet J. Cecil Textbook of Medicine. Philadelphia, PA: W.B. Saunders, 1992. Assar OS, Friedman CM, White RI. The natural history of epistaxis in hereditary hemorrhagic telangiectasia. Laryngoscope 1991; 101: 99780. McCaffrey TV, Kern EB, Lake CE. Management of epistaxis in hereditary hemorrhagic telangiectasia: Review of 80 cases. Arch Otolaryngol 1977; 103: 627-30. Bauer T, Britton P, Lomas D, Wight DG, Friend PJ, Alexander GJ. Liver transplantation for hepatic arteriovenous malformation in hereditary hemorrhagic telangiectasia. J Hepatol 1995; 22: 586-90. Noval J, Nuna F, Anitua MJ, Lopez A, Fuente E, Gallo C. Familial study of patients with hereditary hemorrhagic telangiectasia. An Med Interna 1998; 15: 534-7!
A physicians guide to natural health products that work by james howenstine md, page 223 besides normalizing blood fats, statin drugs such as lipitor atorvastatin ; and pravachol pravastatin ; also reduce inflammation, thus stabilizing any plaques in your coronary arteries and reducing the likelihood of their clotting.
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He noted that they back up a study completed last year, funded by pfizer, showing that lipitor is better than pravachol in limiting the progression of disease in coronary arteries.
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Cell Death. Nuclear staining with Hoechst 33342 Hoechst Pharmaceuticals ; and propidium iodide PI ; was used for morphological.
It is especially important to check with your doctor before combining pravachol with cholestyramine questran ; , cimetidine tagamet ; , colestipol colestid ; , diltiazem cardizem, dilacor, tiazac ; , drugs that suppress the immune system such as sandimmune and neoral ; , erythromycin s and prilosec.
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Bartram CR, de Klein A, Hagemeijer A, et al. Translocation of c-ab1 oncogene correlates with the presence of a Philadelphia chromosome in chronic myelocytic leukaemia. Nature. 1983; 306: 277-280. Ben-Neriah Y, Daley GQ, Mes-Masson AM, et al. The chronic myelogenous leukemia-specific P210 protein is the product of the bcr abl hybrid gene. Science. 1986; 233: 212-214. Burnett AK, Eden OB. The treatment of acute leukaemia. Lancet. 1997; 49: 270-275. Carella AM, Lerma E, Corsetti MT, et al. Autografting with Philadelphia chromosome-negative mobilized hematopoietic progenitor cells in chronic myelogenous leukemia. Blood. 1999; 93: 1534-1539. Carroll M, Ohno-Jones S, Tamura S, et al. CGP 57148, a tyrosine kinase inhibitor, inhibits the growth of cells expressing BCR-ABL, TEL-ABL, and TEL-PDGFR fusion proteins. Blood. 1997; 90: 4947-4952. Chronic Myeloid Leukemia Trialists' Collaborative Group. Interferon alfa versus chemotherapy for chronic myeloid leukemia: a meta-analysis of seven randomized trials. J Natl Cancer Inst. 1997; 89: 1616-1620. Clark SS, McLaughlin J, Crist WM, et al. Unique forms of the abl tyrosine kinase distinguish Ph1-positive CML from Ph1-positive ALL. Science. 1987; 235: 85-88. Coco FL, Nervi C, Avvisati G, et al. Acute promyelocytic leukemia: a curable disease. Leukemia. 1998; 12: 1866-1880. Collins FS, Trent JM. Cancer genetics. In Fauci AS, Braunwald E, Isselbacher KJ, et al. eds. ; . Harrison's Principles of Internal Medicine. New York, McGraw-Hill, 1998: 512-523. Daley GQ, Van Etten RA, Baltimore D. Induction of chronic myelogenous leukemia in mice by the p210bcr abl gene of the Philadelphia chromosome. Science. 1990; 247: 824-830. Derderian PM, Kantarjian HM, Talpaz M, et al. Chronic myelogenous leukemia in the lymphoid blastic phase: characteristics, treatment response, and prognosis. J Med. 1993; 94: 69-74. Druker BJ, Lydon NB. Lessons learned from the development of an Abl tyrosine kinase inhibitor for chronic myelogenous leukemia. J Clin Invest. 2000; 105: 3-7. Faderl S, Kantarjian H, Talpaz M, et al. New treatment approaches for chronic myelogenous leukemia. Semin Oncol. 2000; 27: 578-586, for example, pravachol weight gain.
Cholesterol-lowering statin drugs especially Lescol fight hepatitis C virus, Japanese researchers report. Except for Pravachol, which seems to have no effect on hepatitis C, other statins also have some antihepatitis C activity. But in a new lab test designed to measure the drugs' ability to fight hepatitis C, Lescol was the most potent. Moreover, the test-tube studies show Lescol boosts the effect of alpha interferon, the mainstay of hepatitis C treatment. That's important, since the current combination of long-acting interferon plus ribavirin does not cure some 45% of people infected with the liver-damaging virus and prinivil.
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Fortney J A. The use of hospital resources to treat Incomplete abortions: examples from Latin America. Public Health Rep 1981; 96: 574-79. Misago C, Fonseca W, Correia L etal. Caracterfsticas medicas do aborto provocado entre mulheres admitidas em hospitals de Fortaleza. RevMatem Esc Assis Chateaub 1995: 2: 9-12. Nations MK, Misago C, Fonseca W et al. Women's hidden transcripts about abortion in Brazil. SocSdMcd 1997; 44: 1833-45. Trussell J, Stewart F. The effectiveness of postcoital hormonal contraception. Fam Plann Perspea 1992; 24: 262-64. Trussell J, Stewart F, Guest F, Hatcher RA. Emergency contraceptive pills: a simple proposal to reduce unintended pregnancies. Fam Plann Pcnped 1992; 24: 269-73.
Beginning January 2002, two share ownership plans were introduced for employees of our UK affiliates. The first is the Novartis UK Share Ownership Plan, a UK Inland Revenue-approved plan set up under a Trust. For every two shares purchased, employees will receive one share free. However, the employee would forfeit the matching share and any tax relief received if the employee were to leave the employ of his or her UK employer within 3 years of the award. If the shares are held in the plan for 5 years or more then the employee will not be liable for any form of tax on either the shares they purchased or the free matching shares. The employee's maximum annual investment under this plan is GBP 1, 500. Under the second UK plan, the Novartis UK Incentive Conversion Plan, employees can invest their net incentive bonus, which is the maximum allowable payment to the Novartis UK Share Ownership Plan. For every two shares purchased the employee will receive one free share. But the employee would forfeit the free share if the employee leaves the employ of his or her UK employer within 3 years of the award. Item 7. Major Shareholders and Related Party Transactions 7.A Major Shareholders Based on our share register, we believe that we are not directly or indirectly owned or controlled by another corporation or government, and that there are no arrangements that may result in a change of control. As of December 31, 2004, our registered share capital was CHF 1, 388, 605, 000, divided into 2, 777, 210, 000 shares with a nominal value of CHF 0.50 each. Based on our share register, it appears that approximately 58% of our registered shares are held in Switzerland, and approximately 30% of our shares which are registered by name are held in the United States. However, since certain of our shares are held by brokers or other nominees, and because 24% of our shares are not registered in anyone's name, the above numbers are not representative of the actual number of beneficial owners of our shares located in the US or in Switzerland. As of December 31, 2004 no person or entity was the owner of more than 5% of our shares, whether or not the voting rights of such shares were exercisable. Our largest registered shareholders are Emasan AG 3.2% ; and the Novartis Foundation for Employee Participation 3.1% ; . In 2003, these shareholders held 3.1% and 3.3% respectively. Both shareholders are entered in the share register with voting rights for their entire shareholdings. The largest registered nominee shareholder with voting rights is JPMorgan Chase Bank, N.A. 7.6% ; , which entered into a nominee agreement with us and disclosed the names, addresses and number of shares of the beneficial owners for whose account it holds the shares. JPMorgan Chase Bank, N.A. also holds an additional 7.1% of our shares in its capacity as the Depositary for our ADSs. The second largest nominee shareholder is Nortrust Nominees 2.3% ; . Based on a nominee agreement with us and the regular disclosure of the beneficial owners for whom it holds the shares, this shareholder has voting rights for its entire shareholding No other nominee shareholders nor any beneficial owner known to us holds more than 2% of our shares. Shares We have one class of shares. As of December 31, 2004, a total of 2, 777, 210, 000 shares were issued, with a nominal value of CHF 0.50 each. The shares are fully paid-in and non-assessable. We may issue certificates representing several shares. Shareholders may exchange these certificates at any time for certificates representing smaller numbers of shares, or for individual share certificates. If the owner of the shares consents, we may renounce the printing and delivery of share certificates and promethazine.
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Must recognise that communities are victims; that quality-of-life crime often damages communities more than individuals. If left unaddressed, low-level offences erode communal order, leading to disinvestment and neighbourhood decay and creating an atmosphere where more serious crime can flourish. In order for a community court to help restore the community it should require offenders to compensate neighbourhoods through community service. At the same time, the community should encourage offenders to deal with their individual p ro b ty' ca i o people who break its laws and a form of crime control as supportive initiatives drug treatment, employment and education opportunities etc. - can permanently alter the behaviour of chronic offenders. Advocates of community courts lay great stress on making these services open to all local residents to ensure that the whole community benefits, and to avoid any sense that the offenders are being rewarded for their law-breaking. It is also thought to be important Page 89.
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You are not alone . shift in perceptions about ovarian cancer . Becoming an active partner with your health care team . Making treatment decisions . Learn the language . Making sense of the numbers . Understanding ovarian cancer . Types of ovarian cancer 10 Causes of ovarian cancer risk factors ; 10 Symptoms 11 Diagnosis 11 Surgery and staging 12 Initial therapy for ovarian cancer 15 Risk of recurrence 17 Radiation therapy 17 Intraperitoneal therapy 18 Clinical trials for ovarian cancer 19 Monitoring and follow-up after treatment 20 Timing is everything 23 Treatment for recurrent disease 23 Individualized treatment for recurrent disease 25 Planning the sequence of chemotherapy after recurrence .31 The Patient Active approach to symptom management 32 Managing symptoms and side effects 33 Fatigue 34 Risk of infection 34 Nausea and vomiting 35 Peripheral neuropathy or nerve changes 36 Hair loss alopecia ; 36 Living with ovarian cancer 39 Emotions and ovarian cancer 39 Tools to live by with chronic illness 40 Relationships and intimacy 41 Integrating medicine, mind, and body 42 Healing and hope redefined 42 Resources 43 Important personal information 45 Treatment notes 47 Guide to medical terms 49.
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Many doctors have forgotten this basic tenet of medicine under the hypnotic influence of a blizzard of propaganda from pharmaceutical companies touting their latest patented, money-making, synthetic nightmare.
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Now that Bill S-4, the Senate Tenure Bill, has received second reading and has been referred to the Senate's Legal and Constitutional Affairs Committee, it should receive speedy passage, says Conservative Senator Raynell Andreychuk, who believes that the committee should not duplicate what has already been done at the Special Committee on S-4. "We had a pre-study and a lot of witnesses were heard there. All of their evidence, do we need to restudy it? I would hope not, " Sen. Andreychuk told The Hill Times. "I had great respect for my colleagues who sat on that committee. They did their job adequately, so I would hope that we not start again. We would pick up and take their valuable work and say, what else do we need from a legal and constitutional basis, if we're going to work expeditiously and I trust that we will. I have no reason to doubt that." Sen. Andreychuk said she did not know what the committee will decide, but will recommend that it use all of the evidence from the special committee and all of the speeches that were made on the floor of the Senate to their advantage and not duplicate anything. Liberal Senator Dan Hays said that judging from the speeches made in the Senate, Senators will be interested in bringing some issues up again at the committee. "Senators are still concerned with the constitutional question, " he said. "I'm not sure, but there might be some attention paid to that, although, as you know, the special committee concluded, not unanimously, but concluded in a consensual way that the limiting of Senators' terms falls within the confines of Parliament to deal with." The main focus at committee, however, will be the length of the term, as the Liberals and the Conservatives do not agree on what is acceptable. Liberal Leader Stphane Dion has called for 12-15 year terms, while the bill as it is now stipulates eight years. "The obvious problem that is presented by a Prime Minister who is able to serve two terms and appoint all the senators affecting in a negative way the Parliamentary dynamic of the Senate, " he said. "That needs more attention. So the bill as presented will be looked at with the view of changing that term." Other issues could include the possible reappointment of Senators. While the government consistently said that the Liberal-dominated Senate was delaying a 66-word bill before it received second reading, Sen. Hays said he didn't know how quickly the bill would get through the committee."The committee's work will determine the time frame, " he said. Conservative Senator Don Oliver, former chair of the Legal and Constitutional Affairs committee, agreed however, that the committee was one of the busiest in the Senate and that the work load would determine how fast the bill gets through."There are a number of law and order bills in the commons getting ready to come to the Senate and once they get through the senate, they will go to the committee and so it's going to be incredibly busy. It will receive careful consideration as the other bills, " he said, adding that he supports the bill. "The principle is this. You can become a senator when you're 30 years of age, and you can stay until you're 75. That's 45 years. The question is, if you're unelected and you don't have to account to the general public, is it right that you stay for 45 years? That's the argument that the Prime Minister makes, and I agree with him. It should be a shorter period. The argument before the committee is how long should it be? Should it be eight years, 10 years, 12 years? The said he was open to recommendations from the Senate on that issue and the Senate should hear evidence about length of time. I think it will pass the committee and go back to the Senate for a third reading debate." --by Bea Vongdouangchanh.
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