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Professor HL, UHD, UD, respectively are expected to spend 40% of their time on education and 60% on research and or management. Clinical researchers have also obligations for patient care. For the sake of simplicity a normative approach has been taken to establish input figures appropriately acknowledging that individual variations are common ; . Accordingly, the research input in GUIDE FMS of all selected tenured researchers is set at 40% of their appointment time in the case of non-clinical researchers i.e. generally 0.4 fte ; and 30% in the case of clinical researchers i.e. generally 0.3 fte ; . Non-tenured members of staff, e.g. postdoctoral fellows, are expected to spend 90% and Ph.D. students 70% of their time on research. A.4.2.2 Institutional level The figures for the total research input of the staff of GUIDE FMS in the period 1997-2002 are given in Table 2. A.4.2.3 Program level In table 3 the actually realized research input in fte of research staff members per program is presented for the period 1997-2002. Table 2. Total research input in fte ; of all researchers at GUIDE FMS in the period 19972002. The research input is defined as the actually realized research input see part A.4.2.1 and levodopa, for instance, enalapril maleato.

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Insulin and vanadyl sulfate increase adiponectin cell content through a PI3K-independent and PKB-dependent pathway Andre P. Seale, Lucia A. De Jesus, Min Chul Park and Yangsun Kim Proteonik Inc. Insulin-sensitizing effects of berberine and the extracts of phellodendron amurense cortex in 3T3-L1 adipocytes So-Hui Kim, Eun-Jung Shin and Chang-Kee Hyun Handong Global University Interplay of membrane targeting and dual phosphorylation at S635 and S1179 for the activation of endothelial nitric oxide synthase Yong Chool Boo Kyungpook National University Involvement of ERK1 2 MAPK and S6 kinase in native LDL-induced proliferation of human aortic smooth muscle cells Kyung-Sun Heo, Sungwoo Ryoo, Misun Won, Lila Kim, Xianji Piao, Young-Dae Kim, Seung-Tae Baek, Woo-Jeong Jeong, Jong-Seong Kang * , Dong-Uk Kim and Kwang-Lae Hoe Korea Rescarch Institute of Bioscience and Biotechnology and * Chungnam National University Involvement of Src homology domain-containing tyrosine phosphatase 2 SHP2 ; in H2O2induced JAK-STAT activation in membrane rafts Su Jung Park and Ilo Jou Ajou University Ionizing radiation induces ROS generation via the PI3-kinase-Rac-NADPH oxidase pathway Kyung-Mi Choi, Eun-Sook Cho and Hong-Duck Um Korea Institute of Radiological and Medical Sciences Ionizing radiation kills p53-deficient cells by promoting mitochondrial ROS production in a JNK-dependent manner Hyun Sook Yang and Hong-Duck Um Korea Institute of Radiological and Medical Sciences Isoproterenol modulates the expression of RGS2 in C6 rat glioma cells Sung Dae Kim, Hui Min Lee, Hye Jin Sung, Sun Kyu Park, Tae Wan kim, Kil Soo Kim, Jae Chan Song, 1 2 Seung Chun Park, Won Gi Min Jae Youl Cho and Man Hee Rhee 1 2 Kyungpook National University, Sunchon National University and Kangwon National University JNK is a common mediator of the p53-independent cisplatin cytotoxicity in various cell types: a comparative study with ROS In Hwa Bae and Hong-Duck Um Korea Institute of Radiological and Medical Sciences Junctional membrane inositol 1, 4, 5-trisphosphate receptor complex coordinates sensitization of the silent EGF-induced calcium signaling Eun-Mi Hur, Yong-Soo Park, Yang Hoon Huh * , Seung Hyun Yoo * , Kyung-Chul Woo, Bo-Hwa Choi, and Kyong-Tai Kim Pohang University of Science and Technology and * Inha University Leptin induces, via p38 MAP kinase signal, cell hypertrophy and Hsp27 induction in vascular smooth muscle cells Hye-Jin Shin and Ji Hyung Chung Yonsei University and clarinex. Statement to risks with duricef are fever capoten your provincial carriers.
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61 ; Patent of addition to Application No. : Filed on: 62 ; Divisional to Applcation No.: Filed on: 57 ; Abstract A RAID 3, 4, or 5 ; disk array memory system incorporates a memory system fincorporates a method for the management of faulty data that eliminates the unintentional creation of spurious data when a double fault occurs. For example, when a disk drive failure occurs in one channel and the failed disk is replaced, reconstruction of the data can be achieved because a parity drive channel is provided for correcting errors. If a read error occurs during reconstruction of the failed disk data, the block corresponding to the error block does not allow the reconstruction of the corresponding failed disk block. To prevent the misuse of the two data blocks, a bad data table BDT ; is constructed that lists the addresses of the block just read and the block to be reconstructed. Also a standard filler block is written into the two bad blocks and a new parity block is created. The addresses of all access requests to the memory array are compared with the BDT and, if not listed, the access proceeds. If an address is listed, an error signal is returned. For a listed write request, the bad block address is deleted from the BDT, new data written into the block and a new parity block computed and stored.

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