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Tofranil tofranil tofranil imipramine description manufacturer: novartis chemical name: imipramine tofranil is a tricyclic antidepressant used to treat depression.
18 Vol. 57, No. 1 2006 Northeast Florida Medicine, for example, brand name.

Prescription Medication TOBREX OPH OINT TOBREX OPH SOL Toffranil imipramine ; Tovranil imipramine ; TOFRANIL TB TOFRANIL TB Tolbutamide Topamax Topamax Topamax Topamax TOPAMAX SPRINKLE CAPS TOPAMAX SPRINKLE CAPS Topicort .25% Cr. Topicort .25% Oint TOPICORT GEL TOPICORT MILD CR TOPILENE CREAM W GLYCOL TOPILENE CREAM W GLYCOL TOPILENE LOT W GLYCOL TOPILENE OINT W GLYCOL TOPISALIC LOTION TOPISONE CREAM TOPISONE LOT TOPISONE OINT Topotican Hycamtin ; TOPSYN GEL TORADOL IM AMPUL TORADOL IM AMPUL TORADOL TB TRANDATE TB TRANDATE TB TRANDATE VL Transderm V Patch - OTC TRASICOR TB TRASICOR TB TRASYLOL TRAVATAN OPTH SOL trazodone-generic only trazodone-generic only trazodone-generic only Trental pentoxifylline ; TRIACOMB CREAM TRIADERM CR TRIADERM CR TRIADERM OINT TRIAMCINOLONE ACETONIDE SUSP TRIAMCINOLONE INJ SUSP TRIAMCINOLONE INJ SUSP TRIAMZIDE TRIAVIL TB Triazide - see Dyazide TriCor - see Lipidil Micro or Lipidil Supra. Brummer, "increased drug therapy and indapamide. TERAZOL 3 CREAM 0000MG TERAZOL 7 CREAM 0000MG TERAZOSIN 1MG CAP0001MG TERAZOSIN 2MG CAP0002MG TETRACYCLINE 250M0250MG TETRACYCLINE 500M0500MG THEOPHYLLINE 200M0200MG TIAZAC 120MG CAPS0120MG TIAZAC 180MG CAPS0180MG TIAZAC 240MG CAPS0240MG TIAZAC 300MG CAPS0300MG TIMOLOL 0.5% EYE 0000MG TIMOLOL 0.5% GEL 0000MG TIMOPTIC 0.5% EYE0000MG TIMOPTIC-XE 0.5% 0000MG TOBRADEX EYE DROP0000MG TOBRADEX EYE OINT0000MG TOBRAMYCIN 0.3% E0000MG TOBREX 0.3% EYE D0000MG TOBREX 0.3% EYE O0000MG TOFRANIL 10MG TAB0010MG TOPROL XL 100MG T0100MG TOPROL XL 25MG TA0025MG TOPROL XL 50MG TA0050MG TORADOL 10MG TABL0010MG TRAVATAN 0.004% E0000MG TRAZODONE 100MG T0100MG TRIAMCINOLONE 0.10000MG TRIAMCINOLONE 0.50000MG TRIAMTERENE HCTZ 0000MG TRIAMTERENE HCTZ 0025MG TRIAZOLAM 0.125MG0000MG TRIAZOLAM 0.25MG 0000MG TRICOR 134MG CAPS0134MG TRICOR 200MG CAPS0200MG TRICOR 67MG CAPSU0067MG TRIMOX 125MG 5ML 0125MG TRIMOX 250MG CAPS0250MG TRIMOX 250MG 5ML 0250MG TRIMOX 500MG CAPS0500MG TRIPHASIL-28 TABL0000MG TRIPLE SULFA VAGI0000MG TRIPLE TANNATE PE0000MG TRIVORA -28 TABLET0000MG TUSSI-12 TABLET 0060MG TUSSIONEX PENNKIN0000MG TYLENOL W CODEINE0012MG TYLENOL W CODEINE0030MG TYLENOL W CODEINE0060MG. Introduction . 4 Stories of Victims of Sexual Assault . 5 Facts About Emergency Contraception for Rape Victims. 7 Emergency Contraception Pills and Regimens . 8 Frequently Asked Questions about Hormonal Approaches to EC . Emergency Contraception Practice Guidelines and Policy Statements . 11 Ethical and Religious Directives. 13 Directory of Wisconsin Rape Crisis Centers . 14 Wisconsin SANE Programs. 17 CCRV Wisconsin Resource Guide . 18 Sample Hospital Protocol. 21 Glossary of Terms . 24 and lozol, for instance, xanax. ANTIRETROVIRALS NRTIs- abacavir Ziagen ; , abacavir lamivudine zidovudine Trizivir ; , didanosine ddI, Videx, Videx EC ; , lamivudine Epivir, 3TC ; , lamivudine zidovudine Combivir ; , stavudine d4T, Zerit ; , tenofovir Viread ; , zalcitabine ddC, HIVID ; , zidovudine AZT, Retrovir ; . PIs- amprenavir Agenerase ; , indinavir Crixivan ; , lopinavir ritonavir Kaletra ; , nelfinavir Viracept ; , ritonavir Norvir ; , saquinavir Fortovase ; . NnRTIs- delavirdine Rescriptor ; , efavirenz Sustiva ; , nevirapine Viramune ; . Other- hydroxyurea Hydrea ; . OI DRUGS PHS "A1 OI"s- acyclovir Zovirax ; , azithromycin Zithromax ; , clarithromycin Biaxin ; , famciclovir Famvir ; , fluconazole Diflucan ; , isoniazid Laniazid ; , itraconazole Sporonox ; , pyrazinamide, rifampim Rifadin ; , TMP SMX Bactrim, Septra ; . Other OIs- atovaquone Mepron ; , ciprofloxacin Cipro ; , clindamycin Cleocin ; , clofazimine Lamprene ; , clotrimazole Mycelex ; , dapsone, ethambutol Myambutol ; , ketoconazole Nizoral ; , nystatin Mycostatin ; , metronidazole Flagyl ; , pentamidine Pentam ; , rifabutin Mycobutin ; , valacyclovir Valtrex ; . Hepatitis C- none. TREATMENTS FOR METABOLIC DISORDERS Wasting- megestroll acetate Megace ; . ALL OTHERS alprazolam Xanax ; , amitriptyline Elavil ; , buspirone BuSpar ; , bupropion Weflbutrin ; , carbamazepine Tegretol ; , chlordiazepoxide Librium ; , chlorpromazine Thorazine ; , citalopram Celexa ; , clomipramine Anafrabil ; , clonazepam Klonopin ; , clorazepate Tranxene ; , clozapine Clozaril ; , desipramine Norpramin ; , diazepam Valium ; , doxepin Sinequan ; , droperidol Inapsine ; , estazolam Prosom ; , fluoxetine Prozac ; , fluphenazine Prolixin ; , flurazepam Dalmane ; , fluvoxamine Luvox ; , halazepam Paxipam ; , haloperidol Haldol ; , hydroxyzine Atarax, Vistaril ; , imipramine Tofanil ; , lithium Lithobid ; , lorazepam Ativan ; , loxapine Loxitane ; , mesoridazine Serentil ; , mirtazipine Remeron ; , molindone Moban ; , nefazodone Serzone ; , nortriptyline Pamelor ; , olanzapine Zyprexa ; , oxazepam Serax ; , paroxetine Paxil ; , perphanazine Trilafon ; , pimozide Orap ; , prazepam Centrax ; , prochlorperazine Compazine ; , quetiapine Seroquel ; , risperidone Risperdal ; , sertraline Zoloft ; , temazepam Restoril ; , thioridazine Mellaril ; , thiothixene Navane ; , trazodone Desyrel ; , triazolam Halcion ; , trifluoperazine Stelazine ; , trimipramine Surmontil ; , venlaxafine Effexor ; , zolpidem Ambien.

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Drug names: fluoxetine prozac and others ; , imipramine tofranil and others ; , venlafaxine effexor.
Many tcas of the past now enjoy effective use for sedation as well, including two of the first-imipramine tofranil ; and amitriptyline elavil and isoniazid.
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Home contact advertise the uganda of state for health in charge of primary health care emmanuel otaala, for example, side effects.
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A. All the patients in any given symptom group e.g. women with stress incontinence ; have similar underlying pathophysiology requiring similar treatment, and so urodynamics cannot classify them any better than the symptoms alone. b. Conventional urodynamics does not address the important pathophysiological differences that affect treatment success i.e., we are doing the wrong tests. c. We are doing the right tests, but we are doing them so poorly technically ; that the results are unreliable. d. Current treatments are so non-specific and nonquantitative that the underlying dysfunction is unimportant: treatment works equally well or poorly in any case. Comments: The studies that show that symptoms and urodynamic findings do not correspond very well [1] make possibility "a" unlikely, because they show that similar symptoms have different underlying pathophysiology as revealed by urodynamics. Moreover, we think we know that detrusor overactivity and urge and stress incontinence each come in multiple types. There is strong suspicion, based on expert opinion, that possibility "c" is correct that urodynamics is often done poorly; the International Continence Society and other bodies are improving this situation by establishing standards for proper training and certificate programs in urodynamics. [9] Possibility "b" is a likely explanation: the conventional urodynamic testing that we do is inadequate by itself to unravel the pathophysiological behavior we are interested in. Possibility "d" is also likely to be correct: because of our lack of understanding, current treatments are not aimed accurately at the right target. Some of the mismatch between symptoms and urodynamic findings is due to inherent physiological variability in the function of the lower urinary tract, which is reflected in the urodynamics as well as in the variability of symptoms ; . For example, post-void residual urine in the elderly [10] shows a diurnal variation that implies that there are systematic changes in some unknown controlling parameter. Similarly, analysis of the variability of voiding studies in adult males reveals an unknown source of variation that affects detrusor contractility and urethral resistance simultaneously. [11] Even more and ketorolac.

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Before using ventolin inhalation, tell your doctor if you are taking any of the following medicines: a beta-blocker such as atenolol tenormin ; , metoprolol lopressor, toprol xl ; , propranolol inderal ; , and others; a tricyclic antidepressant such as amitriptyline elavil ; , doxepin sinequan ; , imipramine tofranil ; , nortriptyline pamelor ; , and others; a monoamine oxidase inhibitor maoi ; such as isocarboxazid marplan ; , phenelzine nardil ; , or tranylcypromine parnate another inhaled bronchodilator; or caffeine, diet pills, or decongestants.
Tofranil may be taken with or without food and ketotifen.
Version 1: June 1, 2005 INVASIVE TREATMENT PROTOCOLS T ; EXTERNAL PACEMAKER .T700 TENSION PNEUMOTHORAX DECOMPRESSION .T701 NEEDLE CRICOTHYROTOMY .T702 QUICKTRACH ADDENDUM .T703 INTRAOSSEOUS INFUSION .T704 EMERGENCY USE OF CENTRAL VENOUS ACCESS DEVICE .T705 SPINAL IMMOBILIZATION .T706 NASOTRACHEAL INTUBATION .T708 VERIFICATION OF ENDOTRACHEAL TUBE PLACEMENT T710 APPENDIX A: MEDICATION LIST.APPENDIX A APPENDIX B: PEDIATRIC GUIDELINES.APPENDIX B APPENDIX C: SCOPE OF PRACTICE.APPENDIX C APPENDIX D: MARK-1 KIT PROTOCOL.APPENDIX D APPENDIX E: TRANSPORT OF CONTAMINATED PATIENTS.APPENDIX E APPENDIX F: MANAGEMENT OF MASS CASUALTY INCIDENTS.APPENDIX F APPENDIX G: PEDIATRIC JUMPSTART PROTOCOLS.APPENDIX G. ABSTRACT The human ether-a-go-go-related genes herg ; are expressed in tissues other than heart and brain where the HERG K channels are known to regulate the repolarization of the heart action potential and the neuronal spike-frequency accommodation. We provide evidence that herg1 transcripts are present in human pancreatic islets that were used to study both insulin secretion and electrical activity with radioimmunoassay and single cell perforated patch-clamp techniques, respectively. Glucose- and arginine-induced islets insulin secretion data suggested a net increase of release under perfusion with antiarrhythmic drugs known to selectively block HERG channels. Indeed we could routinely isolate a K current that was recognized as biophysically and pharmacologically similar to the HERG current. An analysis of the glucose- and arginine-induced electrical activity several applications during 30 min ; in terms of firing frequency and putative insulin release was done in control and in the presence of selective blockers of HERG channels: the firing frequency and the release increased by 32% and 77%, respectively. It is concluded that HERG channels have a crucial role in regulating insulin secretion and firing of human -cells. This raises the possibility that some genetically characterized hyperinsulinemic diseases of unknown origin might involve mutations in the HERG channels.--Rosati, B., Marchetti, P., Crociani, O., Lecchi, M., Lupi, R., Arcangeli, A., Olivotto, M., Wanke, E. Glucose- and arginine-induced insulin secretion by human pancreatic -cells: the role of HERG K channels in firing and release. FASEB J. 14, 26012610 2000 and lamictal.

Fluoxetine Prozac ; Amitriptyline Elavil ; Imipramine Torfanil ; Clomipramine Clomicalm, Anafranil ; 1 mg kg PO q 1-3 mg kg PO 2-4 mg kg PO 1-3 mg kg PO 24 hours q 12 hours q 24 hours q 24 hours FIGURE 13-2 Acral Lick Dermatitis. A focal alopecic. LABELER --CHAIN DRUG QUALITEST IVAX PHARMACEUT IVAX PHARMACEUT IVAX PHARMACEUT MAJOR PHARM. MAJOR PHARM. IVAX PHARMACEUT BERGEN BRUNSWIG LEADER --LEADER MEDICINE SHOP MEDICINE SHOP REESE PHARM CO REESE PHARM CO REESE PHARM CO B AUN HI-TECH PHARM. REESE PHARM CO REESE PHARM CO --REESE PHARM CO REESE PHARM CO REESE PHARM CO ALLERGAN INC. ALLERGAN INC. ALLERGAN INC. ALLERGAN INC. ALLERGAN INC. ALLERGAN INC. ALLERGAN INC. --ALLERGAN INC. RUGBY RUGBY RUGBY RUGBY RUGBY RUGBY RUGBY RUGBY ROSS LABS. --IVAX PHARMACEUT IVAX PHARMACEUT NOVO NORDISK-WA NOVO NORDISK-WA NOVO NORDISK-WA and lamotrigine and tofranil, for example, drug interactions.
TABLE 1 Time Frame for Monitoring patient receiving methsuximide Consider doing when patient is displaying toxicity Should be checked every 2 weeks while on the drug. Consider doing when patient is displaying toxicity.
Dosage: 75mg 90; 75mg dragees; 50 dragees; 50 dragees; 50 dragees; 50 dragees; 50 dragees; 30 dragees; 30 3 x 10 ; 25mg tabs; 30 3 x 10 ; 25mg tabs; 28 capsules; 28 capsules; 25mg 90; 25mg dragees; 100 dragees; 100 dragees; 100 dragees; 100 dragees; buy tofranil name toframil mite 20 drg and levothyroxine. I'm disappointed that pfizer is spending millions to convince seniors to pay top dollar for a drug without full disclosure of its effectiveness.
If you recently joined AdvantraRx and learned that AdvantraRx does not cover a drug you were taking when you joined our plan, you are entitled to a one time fill of that prescription up to a 30-day supply. This one time fill will permit up to 30-days of a non-formulary or prior authorized medication to allow you to transition to a formulary medication where appropriate, or to provide time for a formulary exception request. This one time fill will be allowed during the first 90-days of coverage with AdvantraRx. Tofranil side effects common tofrani side effects seen in adults include dizziness, confusion, and dry mouth. Miralles, Agusti Pharmacology & Toxicology Departament J.Uriach & Cia J.Uriach & Cia 08026 Barcelona; Spain pharm uriach, because torfanil indications. Drinking coffee, reading a newspaper, making a phone call, and writing. The third group consisted of activities for which patients made voluntary movements for almost the entire period such as making coffee, walking, setting the table, dressing, etc. The performance of the neural network output was considered to be correct when the neural network gave a value smaller than 0.5 for the 1-minute intervals, which were rated by the physicians with the score 0 no dyskinesia group ; , and when the network gave a score larger than 0.5 for the 1-minute intervals, which were rated by the physicians with a rating 1 or higher dyskinesia group ; . The percentage of correctly classified minutes for the different groups is shown in Table 4. The correct performance of the neural network is between 75% and 100%, depending on the type of movements. The best performance is obtained in the absence of voluntary movements and in the absence of dyskinesia. The network displayed some tendency to erroneously detect absence of dyskinesia in patients with mild dyskinesia who were trying to abstain from any voluntary movements. This is primarily because normal subjects, when sitting in a relaxed position, make small movements with the legs and arms that are hard to distinguish from mild dyskinesia. In general, the neural network was able to correctly distinguish the large majority of LID movements from voluntary movements. A more detailed overview of the rating performance by the neural network for various types of behavior with voluntary movements is shown in Figure 5, which shows the percentage of correctly classified behavior for a selection of activities. In general, approximately 80% of the 1-minute intervals of each activity was correctly classified. Classification algorithms in previous studies showed discrepancies with the rating by physicians for activities with voluntary movements and especially for walking.1315 The neural network gave an extremely well-fit classification for 1-minute intervals of walking for the trunk 100% ; and the leg 96% ; , but less so for the arm 61 and indapamide. The DiFranco Court stated: We disapprove of those cases which have automatically disregarded certain types of evidence merely because it was based upon the plaintiff's subjective complaints or the symptoms of an injury. An expert's diagnosis and the basis for it e.g., the plaintiff's complaints, the physician's observations, and test results ; can be adequately challenged at trial through cross-examination and the presentation of contrary medical evidence. The "serious impairment of body function" threshold requires the plaintiff to prove that his noneconomic losses arose out of a medically identifiable injury which seriously impaired a body function. [DiFranco, supra at 75.].
Adapted with permission by city of toronto: toronto public health.

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If drugs were 100% efficacious, 100% safe, and cost-free, and patients were 100% compliant, the answer would be to treat everyone and early. As this is not the case, the most important factor determining whom and when to treat is an individual's absolute risk of fracture. If the risk is 2 per 1000 women per year, and a drug halves fracture risk, then one event is prevented, one woman will sustain a fracture despite treatment, and 998 who were not going to have a fracture anyhow had treatment. Thus, one fracture is prevented, but 999 women per year are treated without benefit. If the absolute risk is higher, say 2 per 100 women per year, and the drug still halves the risk, again one fracture is prevented, but only 99 women are treated Box 3 ; . Over 3 to 5 years of treatment, this equates to a number needed to treat NNT ; to prevent a fracture of 2025 women, a figure commonly accepted for treatments designed to reduce adverse outcomes in illnesses such as hypertension and hypercholesterolaemia. Hence, cost effectiveness in terms of the NNT to avert one event is mainly driven by the baseline absolute fracture risk. Thus, knowledge of an individual's absolute risk is central to making treatment decisions. The imperative to intervene increases with advancing age, lower bone mineral density BMD ; and previous fracture, as each of these contributes independently to fracture risk. About 85% of fractures occur in women over 60 years of age. An important signal for the need to treat is a prior vertebral or non-vertebral fracture. The risk of further fractures increases threefold to fivefold as the number or severity of prevalent vertebral deformities increases. In a person with osteoporosis, an incident fracture with or without a prevalent fracture at baseline ; increases the absolute risk of a further incident fracture to 30%40% within 3 years.16 Thus, the evidence for anti-fracture efficacy is strongest in patients with a baseline vertebral or nonvertebral fracture. Withholding treatment from these patients does not comply with good medical practice. It is optimal to treat fewer older people over 60 years ; at high risk rather than many younger people at low risk. This ensures that those likely to respond to treatment receive it, and those at low absolute risk, and thus unlikely to benefit, remain untreated. Treatment of the high-risk individual also.
Health care providers must now consider all the evidence, including potential CV risk, when making pharmacotherapy decisions for the prevention of NSAIDinduced gastropathy. Strategies and guidelines have been developed to assist the practitioner in the selection of appropriate pharmacotherapy for patients who require long-term antiinflammatory pharmacotherapy.55-61 The majority of the recommendations are based on the evaluation of patient risk for associated gastropathy. Given the current need to assess for CV risk as well as potential for gastric toxicity, however, the recommendations have evolved. After the withdrawal of rofecoxib and valdecoxib from the US market, and because of concern regarding the previously discussed CV issues with the remaining COX-2 inhibitors, new guidelines have been developed for use when considering long-term therapy with NSAIDs Table 3 ; .59, 61 Prior to the initiation of NSAID therapy in any patient, the option of a non-NSAID should be considered if at all possible to remove the risk of NSAID-induced gastropathy.1, 61 As seen in Table 3, the patient who does not have any significant risk factors for the development of NSAIDassociated complications and does not require cardioprotective aspirin for coronary heart disease CHD ; would be best managed with a traditional NSAID alone. If dyspepsia should develop in this patient, an antacid or antisecretory therapy H2-receptor antagonist or PPI ; could be initiated.60, 61 The patient who has a significant risk eg, history of gastric ulceration, anticoagulation, etc ; of developing NSAIDassociated GI complications but does not take prophylactic aspirin would be. Warren W. Wasiewski, MD. Mayday Pediatric Headache Center. Lancaster, PA As with adults, a complete history should include the historical background of the headache disorder and a thorough description of the headache as it presents now, as many headache disorders evolve over time.1 Once you have obtained the child's headache and medical history, you should have a fairly good understanding of the child's headache disorder. Issues such as time to peak intensity and time to vomiting are helpful in planning treatment. The presence of severe nausea at the onset of headache may alter choice of abortive medications. The general exam should include head circumference and blood pressure. Examination of the skin for neurocutaneous lesions is indicated. The neurologic exam should include visual fields by confrontation and cranial auscultation. In many instances this is the first complete exam the patient has received for this disorder. If the child's exam is normal, as is most often the case, this fact should be conveyed to the family in a very affirmative manner, for example, tofranil elavil. 266 Review articles should consist of an introduction, sections logically titled according to content, and a bibliography. Information on fund-providers and other matters important for the paper e.g. technical assistance ; should be supplied under 'Acknowledgements', which should be placed before references. Tables, graphs, diagrams, figures, etc. should not be enclosed within the body of the text. They may be contained in special files. They should be referred to by type and using Arabic numerals e.g. Table 1: , Figure 1: , etc. ; . The colon should be followed by the text or title. The above data type, numeral and text or title ; should be printed in English and Slovene on separate pages. In the text files they should be placed on separate pages after the body of the text. On the reverse side of these items the name and surname of the first author, the title of the paper, and the name and number of the item should be supplied. Indicate, if necessary, where the item should be placed top, bottom ; . Explanations legends ; should also be both in English and Slovene. All references cited in the text should appear in the Bibliography. They should be numbered in the text in the order in which they appear, marked with Arabic numerals placed in parenthesis. The first reference in the text should determine the number and order of the respective source in the Bibliography. If the author refers again to a source which has already been used in the text, he should cite the number the source had when it was referred to for the first time. Only works which have been published or are available to the public in any other way may be referred to. Unpublished data, unpublished lectures, personal communications and the like should be mentioned in references or notes at the end of the page on which they appear. Sources in the Bibliography should be listed in the order in which they appear in the text. If the source referred to was written by six authors or less, all of them should be cited; in the case of seven or more authors, only the first three should be cited, followed by 'et al.'. Any errata should be submitted to the editor-in-chief in good time after publication so that they may be published in the next issue. EXAMPLES OF REFERENCES Book Hawkins JD. Gene structure and expression. Cambridge: University Press, 1991: 16. Chapter or article in a book Baldessarini RJ. Dopamine receptors and clinical medicine. In Neve KA, Neve RL, eds. The dopamine receptors. Totowa: Human Press, 1996: 475-98. Article in a journal or newspaper Fuji J, Otsu K, Zorzato F, et al. Identification of mutation in porcine ryanodine receptor asociated with malignant hyperthermia. Science 1991; 253: 448-51. Article in proceedings of a meeting or symposium Schnoebelen CS, Louveau I, Bonneau M. Developmental pattern of GH receptor in pig skeletal muscle. In: the 6th Zavrnik memorial meeting. Lipica 1995: 83-6.
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29 table of contents the fda and foreign regulatory authorities impose significant restrictions on the indicated uses and marketing of pharmaceutical products. 1. Advice to women up to age 35 years with a family history of breast cancer should be in keeping with general health advice on the use of the oral contraceptive pill. C ; 2. Women aged over 35 years with a family history of breast cancer should be informed of an increased risk of breast cancer associated with taking the oral contraceptive pill, given that their absolute risk increases with age. C ; 3. For women with BRCA1 mutations, the conflicting effects of a potential increased risk of breast cancer under the age of 40 years and the lifetime protection against ovarian cancer risk from taking the oral contraceptive pill should be discussed. C ; 4. Women should not be prescribed the oral contraceptive pill purely for prevention of cancer, although in some situations reduction in ovarian cancer risk may outweigh any increase in risk of breast cancer. D ; 5. If woman has a BRCA1 mutation and is considering a risk-reducing oophorectomy before the age of 40 years, the oral contraceptive pill should not be prescribed purely for the reduction in ovarian cancer risk. D.

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